From the MetroHealth Medical Center, affiliated with Case Western Reserve University, Cleveland, OH.
J Am Acad Orthop Surg. 2024 Aug 15;32(16):747-753. doi: 10.5435/JAAOS-D-23-00577. Epub 2024 May 8.
The purpose of this study was to report the incidence of iatrogenic sural nerve injury in a large, consecutive sample of surgically managed ankle fractures and to identify factors associated with sural nerve injury and subsequent recovery. We hypothesize that a direct posterior approach may be associated with higher risk of iatrogenic sural nerve injury.
A retrospective cohort study of 265 skeletally mature patients who sustained ankle fractures over a 2-year period was done. All were treated with open reduction and internal fixation of fractured malleoli. Patient, injury, and treatment features were documented. The presence (n = 26, 9.8%) of sural nerve injury and recovery of sural nerve function were noted.
All 26 sural nerve injuries were iatrogenic, occurring postoperatively after open reduction and internal fixation. Patients who sustained sural nerve injuries had more ankle fractures secondary to motor vehicle collisions (23.1% versus 9.2%), more associated trimalleolar fractures (69.2% versus 33.9%), and more Orthopaedic Trauma Association/AO 44B3 fractures (57.7% versus 25.1%), all P < 0.05. A posterior approach to the posterior malleolus through the prone position was used in 20.4% of patients. All 26 of the sural nerve injuries (100%) occurred when the patient was placed prone for a posterior approach, P < 0.001. Therefore, 26 of the 54 patients (48%) treated with a posterior approach sustained an iatrogenic sural nerve injury. 62% of patients had full recovery of sural nerve function with no residual numbness, and patients with nerve recovery had fewer associated fracture-dislocations (23.1% versus 100%, P = 0.003).
A posterior approach for posterior malleolus fixation was associated with a 48% iatrogenic sural nerve injury rate, with 62% recovering full function within 6 months of injury. Morbidity of this approach should be considered, and surgeons should be cautious with nerve handling.
Level III, Therapeutic.
本研究的目的是报告在一个大的连续样本中手术治疗的踝关节骨折中医源性腓肠神经损伤的发生率,并确定与腓肠神经损伤和随后恢复相关的因素。我们假设直接后入路可能与更高的医源性腓肠神经损伤风险相关。
对 2 年内接受踝关节骨折切开复位内固定的 265 例骨骼成熟患者进行回顾性队列研究。所有患者均采用切开复位内固定治疗骨折外踝。记录患者、损伤和治疗特征。注意腓肠神经损伤的存在(n=26,9.8%)和腓肠神经功能的恢复情况。
所有 26 例腓肠神经损伤均为医源性,发生在切开复位内固定术后。发生腓肠神经损伤的患者中,由于机动车事故导致的腓骨骨折更多(23.1%比 9.2%),三踝骨折更多(69.2%比 33.9%),且更符合骨科创伤协会/AO 44B3 骨折(57.7%比 25.1%),所有 P<0.05。通过俯卧位从前向后入路对后踝进行处理的患者占 20.4%。当患者俯卧位进行后入路时,26 例腓肠神经损伤(100%)均发生,P<0.001。因此,54 例采用后入路治疗的患者中有 26 例(48%)发生医源性腓肠神经损伤。62%的患者腓肠神经功能完全恢复,无残留麻木,且神经恢复的患者骨折脱位更少(23.1%比 100%,P=0.003)。
对于后踝固定的后入路与 48%的医源性腓肠神经损伤率相关,62%的患者在损伤后 6 个月内完全恢复功能。应考虑这种方法的发病率,外科医生应谨慎处理神经。
3 级,治疗。