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酷似恶性肿瘤的胆总管良性病变:一种临床常被过度诊断的情况。

Benign Lesion of the Common Bile Duct Mimicking a Malignant Tumor: an Entity Commonly Overdiagnosed Clinically.

作者信息

Osama Md Ali, Dhawan Shashi, Rao Seema, Kumar Arvind, Rawat Saumitra

机构信息

Department of Pathology, Lady Hardinge Medical College, New Delhi, India.

Department of Pathology, Sir Gangaram Hospital, New Delhi, India.

出版信息

Indian J Surg Oncol. 2024 May;15(Suppl 2):331-337. doi: 10.1007/s13193-023-01860-3. Epub 2023 Dec 14.

Abstract

Adenomyomatous hyperplasia and adenomyoma are rare benign inflammatory pseudotumors of the gallbladder arising from Rokitansky-Aschoff sinuses. Occurrence of these hyperplastic conditions in the Vaterian and biliary system is extremely rare and is a concern for gastroenterologists and surgeons in distinguishing them from primary malignancies of the biliary system. Definitive diagnosis by imaging or cytopathological examination is difficult; thus, surgical resection becomes the only choice in such cases to relieve the obstruction. Here, we report two extremely rare cases of adenomyomatous hyperplasia of the extrahepatic bile duct after an extensive diagnostic workup, followed by Whipple's procedure.

摘要

腺肌瘤样增生和腺肌瘤是源于罗-阿窦的罕见的胆囊良性炎性假瘤。这些增生性病变在 Vaterian 和胆道系统中极为罕见,这是胃肠病学家和外科医生在将它们与胆道系统原发性恶性肿瘤区分开来时所关注的问题。通过影像学或细胞病理学检查进行明确诊断很困难;因此,在这种情况下,手术切除成为缓解梗阻的唯一选择。在此,我们报告两例肝外胆管腺肌瘤样增生的极其罕见病例,经过广泛的诊断性检查后,进行了惠普尔手术。

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Adenomyomatosis of the Common Bile Duct and Ampulla of Vater.胆总管及 Vater 壶腹腺肌病
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A case of adenomyomatous hyperplasia of the extrahepatic bile duct.肝外胆管腺肌瘤样增生1例。
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本文引用的文献

1
Unusual cause of a polypoid lesion in the extrahepatic bile duct.肝外胆管息肉样病变的罕见病因。
JGH Open. 2022 Apr 6;6(4):282-284. doi: 10.1002/jgh3.12739. eCollection 2022 Apr.
2
Adenomyomatosis of the Common Bile Duct and Ampulla of Vater.胆总管及 Vater 壶腹腺肌病
GE Port J Gastroenterol. 2021 Feb;28(2):121-133. doi: 10.1159/000507788. Epub 2020 Sep 2.
6
Adenomyomatosis hyperplasia arising in the bile duct.胆管内发生的腺肌增生症
Dig Liver Dis. 2019 Jul;51(7):1060. doi: 10.1016/j.dld.2019.03.006. Epub 2019 Apr 8.

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