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过度抗凝后因高功能甲状腺结节内出血导致的淡漠型甲状腺毒症。

Apathetic thyrotoxicosis due to hemorrhage into a hyperfunctioning thyroid nodule after excessive anticoagulation.

作者信息

Olchovsky D, Pines A, Zwas S T, Itzchak Y, Halkin H

出版信息

South Med J. 1985 May;78(5):609-11. doi: 10.1097/00007611-198505000-00024.

DOI:10.1097/00007611-198505000-00024
PMID:3887580
Abstract

Hyperfunctioning thyroid nodule was diagnosed in a 72-year-old woman. When the nodule reached a diameter of 3.5 cm, overt thyrotoxicosis of the apathetic type was precipitated by hemorrhage into the nodule during a period of excessive anticoagulation. All abnormal physical signs and symptoms as well as laboratory findings returned to normal within a month, without any specific treatment. It was possible to follow this process by repeated B-mode sonographic examinations and thyroid scans.

摘要

一名72岁女性被诊断为功能亢进性甲状腺结节。当结节直径达到3.5厘米时,在抗凝过度期间,结节内出血引发了淡漠型显性甲状腺毒症。所有异常的体征、症状以及实验室检查结果在一个月内恢复正常,未进行任何特殊治疗。通过重复的B超检查和甲状腺扫描能够追踪这一过程。

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Acute airway obstruction due to spontaneous intrathyroid hemorrhage precipitated by anticoagulation therapy.抗凝治疗引发的自发性甲状腺内出血导致急性气道梗阻。
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