[Proteolytic destruction of a hemorrhagic toxin from the venom of the habu (Trimeresurus flavoviridis)].
作者信息
Omori-Satoh T, Sadahiro S, Kondo S
出版信息
Jpn J Med Sci Biol. 1985 Jun;38(3):147-50.
PMID:3906206
Abstract
The major hemorrhagic principle (HR1B) in the venom of Habu (Trimeresurus flavoviridis), a crotalid, was found to be easily inactivated and hydrolyzed by a commercial protease (Nagarse). The ability of the hydrolysate to produce the toxin-neutralizing antibody has been shown to be comparable to or superior to those of two formol toxoids used as the controls, suggesting that inactivation of HR1B by the protease provides a new method for the preparation of Habu toxoid.
摘要
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