自身免疫性“继发性突触病”：N-甲基-D-天冬氨酸受体抗体是否会引发原发性突触外病变？
Autoimmune 'secondary synaptopathies': do NMDAR antibodies cause a primary extra-synaptopathy?
作者信息
Zhao Meng, Lynch David R, Irani Sarosh R
机构信息
Department of Neurology, Mayo Clinic, Jacksonville, FL 32224, USA.
Department of Neuroscience, Mayo Clinic, Jacksonville, FL 32224, USA.
出版信息
Brain. 2024 Aug 1;147(8):2601-2603. doi: 10.1093/brain/awae236.
Abstract
This scientific commentary refers to ‘NMDA receptor autoantibodies primarily impair the extrasynaptic compartment’ by Jamet (https://doi.org/10.1093/brain/awae163).
摘要
本科学评论参考了雅梅特的《NMDA受体自身抗体主要损害突触外区室》（https://doi.org/10.1093/brain/awae163）。
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