Enhancement of Haloperidol-Induced Catalepsy by GPR143, an l-DOPA Receptor, in Striatal Cholinergic Interneurons.
作者信息
Hatzipantelis Cassandra J
机构信息
Institute for Psychedelics and Neurotherapeutics, University of California, Davis, Davis, California 95616
Department of Chemistry, University of California, Davis, Davis, California 95616.
出版信息
J Neurosci. 2024 Aug 14;44(33):e0953242024. doi: 10.1523/JNEUROSCI.0953-24.2024.
Abstract
摘要
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本文引用的文献
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Enhancement of Haloperidol-Induced Catalepsy by GPR143, an L-Dopa Receptor, in Striatal Cholinergic Interneurons.GPR143，一种多巴胺受体，增强纹状体胆碱能中间神经元中海洛因引起的僵住症。
J Neurosci. 2024 Mar 13;44(11):e1504232024. doi: 10.1523/JNEUROSCI.1504-23.2024.
2
Coupling between GPR143 and dopamine D2 receptor is required for selective potentiation of dopamine D2 receptor function by L-3,4-dihydroxyphenylalanine in the dorsal striatum.在背侧纹状体中，L-3,4-二羟基苯丙氨酸对多巴胺D2受体功能的选择性增强需要GPR143与多巴胺D2受体之间的偶联。
J Neurochem. 2023 Apr;165(2):177-195. doi: 10.1111/jnc.15789. Epub 2023 Mar 9.
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Treatment resistant schizophrenia: Clinical, biological, and therapeutic perspectives.治疗抵抗性精神分裂症：临床、生物学和治疗学视角。
Neurobiol Dis. 2019 Nov;131:104257. doi: 10.1016/j.nbd.2018.08.016. Epub 2018 Aug 29.
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