[基因umuC和质粒muc基因在二氧化嘧啶诱导大肠杆菌K12诱变中的作用]
[The role of the gene umuC and plasmid muc genes in mutagenesis induced by dioxidine in E. coli K12].
作者信息
Bakaĭ T S, Khmel' I A, Fonshteĭn L M
出版信息
Mol Gen Mikrobiol Virusol. 1985 Jun(6):33-6.
PMID:3916229
Abstract
The mutability induced by dioxidine in E. coli cells has been shown to be stringently dependent on a function of chromosomal umuC+ gene. Suppression of an umuC mutation by plasmids pKM101 or ColIb, restoring the dioxidine induced mutability, proves the possibility of umuC gene functional complementation by the plasmid muc+ genes.
摘要
已证明二氧嘧啶在大肠杆菌细胞中诱导的突变性严格依赖于染色体umuC⁺基因的功能。质粒pKM101或ColIb对umuC突变的抑制恢复了二氧嘧啶诱导的突变性，这证明了质粒muc⁺基因对umuC基因进行功能互补的可能性。
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