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产生转化生长因子β的调节性T细胞在硬皮病和终末期器官衰竭中的作用。

Role of TGFβ-producing regulatory T cells in scleroderma and end-stage organ failure.

作者信息

Lu Kuo-Cheng, Tsai Kuo-Wang, Hu Wan-Chung

机构信息

Division of Nephrology, Department of Medicine, Taipei Tzu Chi Hospital, Buddhist Tzu Chi Medical Foundation, New Taipei City, Taiwan.

Division of Nephrology, Department of Medicine, Fu Jen Catholic University Hospital, School of Medicine, Fu Jen Catholic University, New Taipei City, Taiwan.

出版信息

Heliyon. 2024 Aug 2;10(15):e35590. doi: 10.1016/j.heliyon.2024.e35590. eCollection 2024 Aug 15.

Abstract

Regulatory T cells (Tregs) are crucial immune cells that initiate a tolerable immune response. Transforming growth factor-beta (TGFβ) is a key cytokine produced by Tregs and plays a significant role in stimulating tissue fibrosis. Systemic sclerosis, an autoimmune disease characterized by organ fibrosis, is associated with an overrepresentation of regulatory T cells. This review aims to identify Treg-dominant tolerable host immune reactions and discuss their association with scleroderma and end-stage organ failure. End-stage organ failures, including heart failure, liver cirrhosis, uremia, and pulmonary fibrosis, are frequently linked to tissue fibrosis. This suggests that TGFβ-producing Tregs are involved in the pathogenesis of these conditions. However, the exact significance of TGFβ and the mechanisms through which it induces tolerable immune reactions during end-stage organ failure remain unclear. A deeper understanding of these mechanisms could lead to improved preventive and therapeutic strategies for these severe diseases.

摘要

调节性T细胞(Tregs)是引发可耐受免疫反应的关键免疫细胞。转化生长因子-β(TGFβ)是Tregs产生的一种关键细胞因子,在刺激组织纤维化中起重要作用。系统性硬化症是一种以器官纤维化为特征的自身免疫性疾病,与调节性T细胞的过度存在有关。本综述旨在确定以Treg为主导的可耐受宿主免疫反应,并讨论它们与硬皮病和终末期器官衰竭的关联。终末期器官衰竭,包括心力衰竭、肝硬化、尿毒症和肺纤维化,常与组织纤维化相关。这表明产生TGFβ的Tregs参与了这些病症的发病机制。然而,TGFβ的确切意义以及它在终末期器官衰竭期间诱导可耐受免疫反应的机制仍不清楚。对这些机制的更深入理解可能会带来针对这些严重疾病的更好的预防和治疗策略。

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