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门静脉高压致脾功能亢进患者多发脾动脉动脉瘤的栓塞治疗：1例报告

Embolization of multiple splenic artery aneurysms in a patient with hypersplenism due to portal hypertension: a case report.

作者信息

Zengo Lucas Victoy Guimarães, Liebich Maria Vitoria Bandeira, Rossi Larissa, Biezus Giuliana Rossato, Toregeani Jeferson Freitas, Park Jong Hun

机构信息

Centro Universitário Fundação Assis Gurgacz - FAG, Cascavel, PR, Brasil.

Faculdade de Ciências Médicas da Santa Casa de São Paulo, São Paulo, SP, Brasil.

出版信息

J Vasc Bras. 2024 Aug 9;23:e20230139. doi: 10.1590/1677-5449.202301392. eCollection 2024.

DOI:10.1590/1677-5449.202301392
PMID:39286298
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11404771/
Abstract

Aneurysms of the splenic artery are the third most common type of intra-abdominal aneurysms and the most common type of visceral aneurysms. Portal hypertension is a significant risk factor for development of these aneurysms. We report the case of a white, female, 52-year-old patient with multiple splenic artery aneurysms and hypersplenism secondary to portal hypertension and cirrhosis. Abdominal angiotomography identified six splenic aneurysms. In this scenario, an endovascular intervention was scheduled to conduct embolization using controlled release coils and Onyx™ embolization agent. The three largest aneurysms were treated. Control angiographs showed good exclusion of the aneurysms. The endovascular technique therefore proved to be a good choice considering the patient's comorbidities and blood disorders. In this case, the procedure was successful. There were no immediate or long-term complications. The patient recovered well and is in clinical follow-up.

摘要

脾动脉瘤是腹腔内动脉瘤的第三大常见类型，也是内脏动脉瘤最常见的类型。门静脉高压是这些动脉瘤形成的重要危险因素。我们报告了一例52岁白人女性患者，患有多发脾动脉瘤，并继发于门静脉高压和肝硬化的脾功能亢进。腹部血管造影术发现了6个脾动脉瘤。在这种情况下，计划进行血管内介入治疗，使用可控释放线圈和Onyx™栓塞剂进行栓塞。治疗了三个最大的动脉瘤。对照血管造影显示动脉瘤得到了良好的封堵。考虑到患者的合并症和血液疾病，血管内技术因此被证明是一个不错的选择。在该病例中，手术成功。没有即刻或长期并发症。患者恢复良好，正在接受临床随访。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2a8e/11404771/3c6fd25ac4e8/jvb-23-e20230139-g04.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2a8e/11404771/9e1cf77a7dd5/jvb-23-e20230139-g01-en.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2a8e/11404771/f9dbb4ddff66/jvb-23-e20230139-g02-en.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2a8e/11404771/7ffda217463e/jvb-23-e20230139-g03-en.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2a8e/11404771/3c6fd25ac4e8/jvb-23-e20230139-g04-en.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2a8e/11404771/9e1cf77a7dd5/jvb-23-e20230139-g01.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2a8e/11404771/f9dbb4ddff66/jvb-23-e20230139-g02.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2a8e/11404771/7ffda217463e/jvb-23-e20230139-g03.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2a8e/11404771/3c6fd25ac4e8/jvb-23-e20230139-g04.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2a8e/11404771/9e1cf77a7dd5/jvb-23-e20230139-g01-en.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2a8e/11404771/f9dbb4ddff66/jvb-23-e20230139-g02-en.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2a8e/11404771/7ffda217463e/jvb-23-e20230139-g03-en.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2a8e/11404771/3c6fd25ac4e8/jvb-23-e20230139-g04-en.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2a8e/11404771/9e1cf77a7dd5/jvb-23-e20230139-g01.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2a8e/11404771/f9dbb4ddff66/jvb-23-e20230139-g02.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2a8e/11404771/7ffda217463e/jvb-23-e20230139-g03.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/2a8e/11404771/3c6fd25ac4e8/jvb-23-e20230139-g04.jpg

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Multiple Giant Splenic Artery Aneurysms Causing Sinistral (Left-Sided) Portal Hypertension.
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