肠外营养期间肝损伤的发病机制：脂褐素是线索吗？
Pathogenesis of liver damage during parenteral nutrition: is lipofuscin a clue?
作者信息
Berger H M, Den Ouden A L, Calame J J
出版信息
Arch Dis Child. 1985 Aug;60(8):774-6. doi: 10.1136/adc.60.8.774.
Abstract
Lipofuscin develops in cells when peroxidation damage occurs. Its development in the liver of patients receiving prolonged parenteral nutrition suggests that peroxidation damage by free radicals has occurred. Deficiencies in antioxidants such as vitamin E may be an important contributing factor.
摘要
当发生过氧化损伤时，脂褐素在细胞中形成。其在接受长期肠外营养患者肝脏中的出现表明自由基造成了过氧化损伤。维生素E等抗氧化剂缺乏可能是一个重要的促成因素。
Zotero 插件