Takin' Out the Trash for Brain Health!
作者信息
Varvel Nicholas H
机构信息
Department of Pharmacology and Chemical Biology, Emory School of Medicine.
出版信息
Epilepsy Curr. 2024 May 16;24(4):292-294. doi: 10.1177/15357597241253503. eCollection 2024 Jul-Aug.
Abstract
摘要
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Epilepsy Curr. 2024 May 16;24(4):292-294. doi: 10.1177/15357597241253503. eCollection 2024 Jul-Aug.
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本文引用的文献
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Immunoproteasome deficiency results in age-dependent development of epilepsy.免疫蛋白酶体缺陷导致癫痫随年龄发展。
Brain Commun. 2024 Jan 29;6(1):fcae017. doi: 10.1093/braincomms/fcae017. eCollection 2024.
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The proteasome: A key modulator of nervous system function, brain aging, and neurodegenerative disease.蛋白酶体：神经系统功能、大脑衰老和神经退行性疾病的关键调节因子。
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TAU ablation in excitatory neurons and postnatal TAU knockdown reduce epilepsy, SUDEP, and autism behaviors in a Dravet syndrome model.在 Dravet 综合征模型中，兴奋性神经元 TAU 消融和出生后 TAU 敲低可减少癫痫、SUDEP 和自闭症行为。
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The Function of Immunoproteasomes-An Immunologists' Perspective.免疫蛋白酶体的功能——免疫学家的视角。
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The immunoproteasome β5i subunit is a key contributor to ictogenesis in a rat model of chronic epilepsy.免疫蛋白酶体β5i 亚基是慢性癫痫大鼠模型致痫发作的关键贡献者。
Brain Behav Immun. 2015 Oct;49:188-96. doi: 10.1016/j.bbi.2015.05.007. Epub 2015 Jun 1.
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Tau loss attenuates neuronal network hyperexcitability in mouse and Drosophila genetic models of epilepsy.tau 蛋白缺失可减轻癫痫的小鼠和果蝇遗传学模型中的神经元网络过度兴奋。
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Mice completely lacking immunoproteasomes show major changes in antigen presentation.缺乏免疫蛋白酶体的小鼠在抗原呈递方面表现出重大变化。
Nat Immunol. 2011 Dec 25;13(2):129-35. doi: 10.1038/ni.2203.
8
Post-treatment, but not pre-treatment, with the selective cyclooxygenase-2 inhibitor celecoxib markedly enhances functional recovery from kainic acid-induced neurodegeneration.用选择性环氧化酶-2抑制剂塞来昔布进行治疗后（而非治疗前），可显著增强由海藻酸诱导的神经变性后的功能恢复。
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Nimesulide aggravates kainic acid-induced seizures in the rat.尼美舒利会加重大鼠由海藻酸诱发的癫痫发作。
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