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Triamcinolone Acetonide Protects Against Light-Induced Retinal Degeneration by Activating Anti-Inflammatory STAT6/Arg1 Signaling in Microglia.

作者信息

Tang Xiangcheng, Liu Wei, Liang Jia, Zhu Xingfei, Ge Xiangyu, Fang Dong, Ling Lirong, Yuan Fanglan, Zeng Kun, Chen Qingshan, Zhang Guoming, Gong Lili, Zhang Shaochong

机构信息

Shenzhen Eye Hospital, Shenzhen Eye Institute, JinanUniversity, 18 Zetian Road, Shenzhen, 518040, Guangdong, China.

State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-Sen University, Guangzhou, 510060, Guangdong, China.

出版信息

Inflammation. 2024 Sep 28. doi: 10.1007/s10753-024-02152-w.

DOI:10.1007/s10753-024-02152-w
PMID:39340587
Abstract
摘要

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Triamcinolone Acetonide Protects Against Light-Induced Retinal Degeneration by Activating Anti-Inflammatory STAT6/Arg1 Signaling in Microglia.曲安奈德通过激活小胶质细胞中的抗炎STAT6/精氨酸酶1信号通路来预防光诱导的视网膜变性。
Inflammation. 2024 Sep 28. doi: 10.1007/s10753-024-02152-w.
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Correction to: Triamcinolone Acetonide Protects Against Light-Induced Retinal Degeneration by Activating Anti-Inflammatory STAT6/Arg1 Signaling in Microglia.对《曲安奈德通过激活小胶质细胞中的抗炎STAT6/精氨酸酶1信号通路预防光诱导的视网膜变性》一文的修正
Inflammation. 2024 Dec 27. doi: 10.1007/s10753-024-02224-x.
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Polyamidoamine dendrimer-conjugated triamcinolone acetonide attenuates nerve injury-induced spinal cord microglia activation and mechanical allodynia.聚酰胺-胺树枝状大分子共轭曲安奈德可减轻神经损伤诱导的脊髓小胶质细胞活化和机械性异常性疼痛。
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STAT6/Arg1 promotes microglia/macrophage efferocytosis and inflammation resolution in stroke mice.STAT6/Arg1 促进中风小鼠小胶质细胞/巨噬细胞的吞噬作用和炎症消退。
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本文引用的文献

1
Arginase-1 promotes lens epithelial-to-mesenchymal transition in different models of anterior subcapsular cataract.精氨酸酶-1 在前囊下白内障不同模型中促进晶状体上皮细胞向间充质细胞转化。
Cell Commun Signal. 2023 Sep 18;21(1):236. doi: 10.1186/s12964-023-01210-4.
2
The BET PROTAC inhibitor dBET6 protects against retinal degeneration and inhibits the cGAS-STING in response to light damage.BET 蛋白PROTAC 抑制剂 dBET6 可防止视网膜变性，并抑制光损伤后的 cGAS-STING 反应。
J Neuroinflammation. 2023 May 22;20(1):119. doi: 10.1186/s12974-023-02804-y.
3
JAK: Not Just Another Kinase.
JAK：不只是另一种激酶。
Mol Cancer Ther. 2022 Dec 2;21(12):1757-1764. doi: 10.1158/1535-7163.MCT-22-0323.
4
Genetic targeting or pharmacological inhibition of galectin-3 dampens microglia reactivity and delays retinal degeneration.基因靶向或半乳糖凝集素-3 的药理学抑制可抑制小胶质细胞的反应性并延缓视网膜变性。
J Neuroinflammation. 2022 Sep 17;19(1):229. doi: 10.1186/s12974-022-02589-6.
5
The matricellular protein SPARC induces inflammatory interferon-response in macrophages during aging.基质细胞蛋白 SPARC 在衰老过程中诱导巨噬细胞产生炎症性干扰素反应。
Immunity. 2022 Sep 13;55(9):1609-1626.e7. doi: 10.1016/j.immuni.2022.07.007. Epub 2022 Aug 12.
6
Interleukin-4 promotes microglial polarization toward a neuroprotective phenotype after retinal ischemia/reperfusion injury.白细胞介素-4促进视网膜缺血/再灌注损伤后小胶质细胞向神经保护表型极化。
Neural Regen Res. 2022 Dec;17(12):2755-2760. doi: 10.4103/1673-5374.339500.
7
Multinucleated Retinal Pigment Epithelial Cells Adapt to Vision and Exhibit Increased DNA Damage Response.多核视网膜色素上皮细胞适应视觉并表现出增强的 DNA 损伤反应。
Cells. 2022 May 5;11(9):1552. doi: 10.3390/cells11091552.
8
Inhibition of cGAS-STING by JQ1 alleviates oxidative stress-induced retina inflammation and degeneration.JQ1 通过抑制 cGAS-STING 减轻氧化应激诱导的视网膜炎症和变性。
Cell Death Differ. 2022 Sep;29(9):1816-1833. doi: 10.1038/s41418-022-00967-4. Epub 2022 Mar 28.
9
Microglia: Immune and non-immune functions.小胶质细胞：免疫和非免疫功能。
Immunity. 2021 Oct 12;54(10):2194-2208. doi: 10.1016/j.immuni.2021.09.014.
10
Rapid morphologic changes to microglial cells and upregulation of mixed microglial activation state markers induced by P2X7 receptor stimulation and increased intraocular pressure.P2X7 受体刺激和眼内压升高诱导小胶质细胞形态的快速变化和混合小胶质细胞激活状态标志物的上调。
J Neuroinflammation. 2021 Sep 20;18(1):217. doi: 10.1186/s12974-021-02251-7.