Bharati S, Lev M
J Thorac Cardiovasc Surg. 1986 Jan;91(1):79-85.
Two hundred nineteen hearts with preductal coarctation (tubular hypoplasia) were analyzed in the laboratory to aid in surgical correction. One hundred fourteen were without and 105 were with ventricular septal defect. We believe the following anatomic findings may be responsible for the mortality and morbidity in surgical repair. The smallness and thinness of the left atrium (40% in those hearts without and 24% in those with ventricular septal defect); the smallness and thinness of the left ventricle (46% in those without and 32% in those with ventricular septal defect); the presence of mitral stenosis (35% in those without and 24% in those with ventricular septal defect); the presence of unicuspid or bicuspid dysplastic aortic valve (49% in those without and 50% in those with ventricular septal defect); the presence of a combination of aortic and mitral stenosis (22% in those without and 9% in those with ventricular septal defect); the location of the ventricular septal defect when present; and the possibility of stenosis or insufficiency of the dysplastic aortic valve in the late postoperative period.
在实验室对219例导管前型主动脉缩窄(管状发育不全)的心脏进行了分析,以辅助手术矫正。114例无室间隔缺损,105例有室间隔缺损。我们认为以下解剖学发现可能是手术修复中死亡率和发病率的原因。左心房小而薄(无室间隔缺损的心脏中占40%,有室间隔缺损的心脏中占24%);左心室小而薄(无室间隔缺损的心脏中占46%,有室间隔缺损的心脏中占32%);存在二尖瓣狭窄(无室间隔缺损的心脏中占35%,有室间隔缺损的心脏中占24%);存在单瓣或二瓣发育异常的主动脉瓣(无室间隔缺损的心脏中占49%,有室间隔缺损的心脏中占50%);存在主动脉和二尖瓣狭窄的组合(无室间隔缺损的心脏中占22%,有室间隔缺损的心脏中占9%);室间隔缺损存在时的位置;以及术后晚期发育异常的主动脉瓣狭窄或关闭不全的可能性。