[实验性惊厥时大鼠脑线粒体对琥珀酸的氧化作用]
[Succinate oxidation by rat brain mitochondria in experimental convulsions].
作者信息
Khazanov V A, Zimina T A, Saratikov A S
出版信息
Biull Eksp Biol Med. 1986 Jan;101(1):35-8.
PMID:3942813
Abstract
Corasole-induced convulsive fits are accompanied by the activation of succinate oxidation in the isolated mitochondria, paralleled by the mounting effect of factors limiting succinate dehydrogenase activity. Diverse seasonal sensitivity to corasole correlates with the inhibition of succinate-dependent energy supply for the functional neuronal activity.
摘要
可拉索诱发的惊厥发作伴随着分离线粒体中琥珀酸氧化的激活，同时限制琥珀酸脱氢酶活性的因素的作用也在增强。对可拉索的不同季节敏感性与功能性神经元活动中琥珀酸依赖性能量供应的抑制相关。
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