Author Correction: NOTCH localizes to mitochondria through the TBC1D15-FIS1 interaction and is stabilized via blockade of E3 ligase and CDK8 recruitment to reprogram tumor-initiating cells.
作者信息
Choi Hye Yeon, Zhu Yicheng, Zhao Xuyao, Mehta Simran, Hernandez Juan Carlos, Lee Jae-Jin, Kou Yi, Machida Risa, Giacca Mauro, Del Sal Giannino, Ray Ratna, Eoh Hyungjin, Tahara Stanley M, Chen Lin, Tsukamoto Hidekazu, Machida Keigo
机构信息
Departments of Molecular Microbiology and Immunology, University of Southern California, Los Angeles, CA, USA.
Viterbi School of Engineering, University of Southern California, Los Angeles, CA, USA.
出版信息
Exp Mol Med. 2024 Oct;56(10):2323. doi: 10.1038/s12276-024-01325-9.
Abstract
摘要
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1
Author Correction: NOTCH localizes to mitochondria through the TBC1D15-FIS1 interaction and is stabilized via blockade of E3 ligase and CDK8 recruitment to reprogram tumor-initiating cells.作者更正：NOTCH通过TBC1D15-FIS1相互作用定位于线粒体，并通过阻断E3连接酶和CDK8募集而稳定，从而对肿瘤起始细胞进行重编程。
Exp Mol Med. 2024 Oct;56(10):2323. doi: 10.1038/s12276-024-01325-9.
2
NOTCH localizes to mitochondria through the TBC1D15-FIS1 interaction and is stabilized via blockade of E3 ligase and CDK8 recruitment to reprogram tumor-initiating cells.NOTCH 通过 TBC1D15-FIS1 相互作用定位于线粒体，并通过阻断 E3 连接酶和 CDK8 的募集来稳定，从而重编程肿瘤起始细胞。
Exp Mol Med. 2024 Feb;56(2):461-477. doi: 10.1038/s12276-024-01174-6. Epub 2024 Feb 27.
3
A conserved, noncanonical insert in FIS1 mediates TBC1D15 and DRP1 recruitment for mitochondrial fission.一个保守的、非规范的 FIS1 插入片段介导 TBC1D15 和 DRP1 招募以进行线粒体分裂。
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4
Fis1 acts as a mitochondrial recruitment factor for TBC1D15 that is involved in regulation of mitochondrial morphology.Fis1 作为 TBC1D15 的线粒体募集因子，参与调节线粒体形态。
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5
TBC1D15/RAB7-regulated mitochondria-lysosome interaction confers cardioprotection against acute myocardial infarction-induced cardiac injury.TBC1D15/RAB7调节的线粒体-溶酶体相互作用赋予对急性心肌梗死所致心脏损伤的心脏保护作用。
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TBC1D15-Drp1 interaction-mediated mitochondrial homeostasis confers cardioprotection against myocardial ischemia/reperfusion injury.TBC1D15与动力相关蛋白1（Drp1）相互作用介导的线粒体稳态赋予心肌对缺血/再灌注损伤的保护作用。
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7
Biogenic selenium nanoparticles alleviate intestinal barrier injury in mice through TBC1D15/Fis1/Rab7 pathway.生物源硒纳米颗粒通过 TBC1D15/Fis1/Rab7 通路减轻小鼠肠道屏障损伤。
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8
p53 destabilizing protein skews asymmetric division and enhances NOTCH activation to direct self-renewal of TICs.p53 不稳定蛋白使不对称分裂倾斜，并增强 NOTCH 激活，以指导 TIC 的自我更新。
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9
Structural studies of human fission protein FIS1 reveal a dynamic region important for GTPase DRP1 recruitment and mitochondrial fission.人类裂变蛋白 FIS1 的结构研究揭示了一个对于 GTPase DRP1 招募和线粒体裂变很重要的动态区域。
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10
Mid51/Fis1 mitochondrial oligomerization complex drives lysosomal untethering and network dynamics.Mid51/Fis1线粒体寡聚化复合物驱动溶酶体解栓及网络动态变化。
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