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The COVID-19 thrombus: distinguishing pathological, mechanistic, and phenotypic features and management.

作者信息

Del Carpio-Orantes Luis

机构信息

Instituto Mexicano del Seguro Social, Mexico City, Mexico.

出版信息

J Thromb Thrombolysis. 2025 Jan;58(1):161. doi: 10.1007/s11239-024-03056-0. Epub 2024 Nov 4.

DOI:10.1007/s11239-024-03056-0
PMID:39496847
Abstract
摘要

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本文引用的文献

1
Intermediate dose enoxaparin in hospitalized patients with moderate-severe COVID-19: a pilot phase II single-arm study, INHIXACOVID19.住院的中重度 COVID-19 患者中中等剂量依诺肝素：一项 INHIXACOVID19 的 II 期单臂先导研究。
BMC Infect Dis. 2023 Oct 24;23(1):718. doi: 10.1186/s12879-023-08297-7.
2
[Intertwined long COVID theories to explain its etiopathogenesis].[解释长新冠发病机制的相互交织理论]
Rev Med Inst Mex Seguro Soc. 2023 May 2;61(3):256-257.
3
Increased Levels of Inflammatory and Endothelial Biomarkers in Blood of Long COVID Patients Point to Thrombotic Endothelialitis.
长新冠患者血液中炎症和血管生物标志物水平升高，提示存在血栓性血管炎。
Semin Thromb Hemost. 2024 Mar;50(2):288-294. doi: 10.1055/s-0043-1769014. Epub 2023 May 19.
4
Relative Hypercoagulopathy of the SARS-CoV-2 Beta and Delta Variants when Compared to the Less Severe Omicron Variants Is Related to TEG Parameters, the Extent of Fibrin Amyloid Microclots, and the Severity of Clinical Illness.与严重程度较轻的奥密克戎变异株相比，SARS-CoV-2 的贝塔和德尔塔变异株存在相对高凝状态，这与 TEG 参数、纤维蛋白淀粉样微栓子的程度以及临床疾病的严重程度有关。
Semin Thromb Hemost. 2022 Oct;48(7):858-868. doi: 10.1055/s-0042-1756306. Epub 2022 Sep 29.
5
Proteomics of fibrin amyloid microclots in long COVID/post-acute sequelae of COVID-19 (PASC) shows many entrapped pro-inflammatory molecules that may also contribute to a failed fibrinolytic system.长新冠/COVID-19 后急性后遗症（PASC）中纤维蛋白原纤维微血栓的蛋白质组学研究表明，其中有许多被捕获的促炎分子，这也可能导致纤溶系统失效。
Cardiovasc Diabetol. 2022 Sep 21;21(1):190. doi: 10.1186/s12933-022-01623-4.