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创伤性截瘫后脊髓拴系伴迟发性神经功能恶化。

Spinal cord tethering after traumatic paraplegia with late neurological deterioration.

作者信息

Ragnarsson T S, Durward Q J, Nordgren R E

出版信息

J Neurosurg. 1986 Mar;64(3):397-401. doi: 10.3171/jns.1986.64.3.0397.

Abstract

The case of a 49-year-old man with traumatic complete T-5 paraplegia of 30 years' duration is presented. Over the last 10 years, he developed cervical myelopathy suggestive of syringomyelia. Work-up, including metrizamide myelography and delayed computerized tomography scanning, revealed an atrophic cord without a syrinx, and a complete block to contrast material at T-5. At operation, the cord was found to be tethered at the original injury site, and was released by transection. This resulted in immediate improvement in the somatosensory evoked potentials and marked postoperative clinical improvement. It is postulated that cord tethering can cause delayed myelopathy in patients with traumatic cord injury. Release of the tethered cord should be considered if a posttraumatic syrinx is not found.

摘要

本文报告了一例49岁男性患者,其因创伤导致T-5完全性截瘫已达30年。在过去10年中,他出现了提示脊髓空洞症的颈髓病。包括甲泛葡胺脊髓造影和延迟计算机断层扫描在内的检查显示脊髓萎缩但无空洞形成,且在T-5水平造影剂完全受阻。手术时发现脊髓在原损伤部位被束缚,通过横断将其松解。这使得体感诱发电位立即改善,术后临床症状也有显著改善。据推测,脊髓束缚可导致创伤性脊髓损伤患者出现延迟性脊髓病。如果未发现创伤后脊髓空洞症,应考虑松解束缚的脊髓。

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