Reply to Pomara et al: A potential role for sTREM2 in PTSD?
作者信息
Bonomi Robin, Pietrzak Robert H, Cosgrove Kelly P
机构信息
Department of Psychiatry, Yale University School of Medicine, New Haven, CT 06511.
出版信息
Proc Natl Acad Sci U S A. 2024 Nov 26;121(48):e2418104121. doi: 10.1073/pnas.2418104121. Epub 2024 Nov 13.
Abstract
摘要
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Reply to Pomara et al: A potential role for sTREM2 in PTSD?对波马拉等人的回复：可溶性触发受体表达分子2（sTREM2）在创伤后应激障碍（PTSD）中可能发挥的作用？
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本文引用的文献
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Microglia-mediated neuroimmune suppression in posttraumatic stress disorder.小胶质细胞介导的创伤后应激障碍中的神经免疫抑制
Proc Natl Acad Sci U S A. 2024 Nov 26;121(48):e2417472121. doi: 10.1073/pnas.2417472121. Epub 2024 Nov 13.
2
Microglia-mediated neuroimmune suppression in PTSD is associated with anhedonia.创伤后应激障碍中小胶质细胞介导的神经免疫抑制与快感缺失有关。
Proc Natl Acad Sci U S A. 2024 Aug 27;121(35):e2406005121. doi: 10.1073/pnas.2406005121. Epub 2024 Aug 22.
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Microglia states and nomenclature: A field at its crossroads.小胶质细胞状态和命名：一个处于十字路口的领域。
Neuron. 2022 Nov 2;110(21):3458-3483. doi: 10.1016/j.neuron.2022.10.020.
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sTREM-1 promotes the phagocytic function of microglia to induce hippocampus damage via the PI3K-AKT signaling pathway.sTREM-1 通过 PI3K-AKT 信号通路促进小胶质细胞的吞噬功能，从而诱导海马损伤。
Sci Rep. 2022 Apr 29;12(1):7047. doi: 10.1038/s41598-022-10973-8.
5
Soluble TREM2: Innocent bystander or active player in neurological diseases?可溶性 TREM2：神经疾病中的无辜旁观者还是活跃参与者？
Neurobiol Dis. 2022 Apr;165:105630. doi: 10.1016/j.nbd.2022.105630. Epub 2022 Jan 15.
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From psychiatry to neurology: Psychedelics as prospective therapeutics for neurodegenerative disorders.从精神病学到神经科学：迷幻剂作为神经退行性疾病的潜在治疗方法。
J Neurochem. 2022 Jul;162(1):89-108. doi: 10.1111/jnc.15509. Epub 2021 Oct 22.
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Longitudinal biomarkers in amyotrophic lateral sclerosis.肌萎缩侧索硬化症的纵向生物标志物。
Ann Clin Transl Neurol. 2020 Jul;7(7):1103-1116. doi: 10.1002/acn3.51078. Epub 2020 Jun 9.
8
Recent Developments in TSPO PET Imaging as A Biomarker of Neuroinflammation in Neurodegenerative Disorders.TSPO PET 成像作为神经退行性疾病神经炎症生物标志物的最新进展。
Int J Mol Sci. 2019 Jun 28;20(13):3161. doi: 10.3390/ijms20133161.
9
Early increase of CSF sTREM2 in Alzheimer's disease is associated with tau related-neurodegeneration but not with amyloid-β pathology.阿尔茨海默病患者脑脊液 sTREM2 的早期增加与 tau 相关的神经退行性变有关，但与淀粉样蛋白-β 病理学无关。
Mol Neurodegener. 2019 Jan 10;14(1):1. doi: 10.1186/s13024-018-0301-5.
10
The Microglial Innate Immune Receptor TREM2 Is Required for Synapse Elimination and Normal Brain Connectivity.小胶质细胞先天免疫受体 TREM2 对于突触消除和正常大脑连接是必需的。
Immunity. 2018 May 15;48(5):979-991.e8. doi: 10.1016/j.immuni.2018.04.016. Epub 2018 May 8.
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