Maeda H, Nakao H
Physiol Behav. 1986;36(2):339-42. doi: 10.1016/0031-9384(86)90026-0.
Effects of amygdaloid lesions on the switch-off behavior (SOB) and behavioral changes induced by a delayed reinforcement (DR) for SOB were investigated in 12 cats. The DR consisted of putting a delay time of 1.6 or 2.4 sec between a plate-pushing response and the actual termination of hypothalamic stimulation, and it produced a marked prolongation of response time for SOB (delay phenomenon). Bilateral electrolytic lesions of the amygdala had only limited effects on the SOB and the delay phenomenon; they tended to enhance the prolongation of response time induced by DR in 6 cats and it was accompanied by mild prolongation of response time of subsequent immediate reinforcement trials. These effects were interpreted to be due to apparent attenuation of emotion-producing potency of the hypothalamic stimulation, induced by reduction of facilitatory environmental influences on it.
在12只猫身上研究了杏仁核损伤对切断行为（SOB）以及由SOB的延迟强化（DR）所诱导的行为变化的影响。DR包括在推板反应与下丘脑刺激实际终止之间设置1.6秒或2.4秒的延迟时间，并且它导致SOB的反应时间显著延长（延迟现象）。杏仁核的双侧电解损伤对SOB和延迟现象的影响有限；它们倾向于增强6只猫中由DR诱导的反应时间延长，并且随后的即时强化试验的反应时间也伴有轻度延长。这些影响被解释为是由于对下丘脑刺激的促进性环境影响减少，导致其产生情绪的效力明显减弱所致。
