Correction: Bone marrow mesenchymal stem cells expressing Neat-1, Hotair-1, miR-21, miR-644, and miR-144 subsided cyclophosphamide-induced ovarian insufficiency by remodeling the IGF-1-kisspeptin system, ovarian apoptosis, and angiogenesis.
作者信息
Ahmed Amany I, Dowidar Mohamed F, Negm Asmaa F, Abdellatif Hussein, Alanazi Asma, Alassiri Mohammed, Samy Walaa, Mekawy Dina Mohamed, Abdelghany Eman M A, El-Naseery Nesma I, Ibrahem Mohamed A, Albadawi Emad Ali, Salah Wed, Eldesoqui Mamdouh, Tîrziu Emil, Bucur Iulia Maria, Arisha Ahmed Hamed, Khamis Tarek
机构信息
Department of Biochemistry, Faculty of Veterinary Medicine, Zagazig University, Zagazig, 44519, Egypt.
Department of Human and Clinical Anatomy, College of Medicine and Health Sciences, Sultan Qaboos University, Muscat, Sultanate of Oman.
出版信息
J Ovarian Res. 2024 Nov 29;17(1):239. doi: 10.1186/s13048-024-01564-4.
Abstract
摘要
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1
Correction: Bone marrow mesenchymal stem cells expressing Neat-1, Hotair-1, miR-21, miR-644, and miR-144 subsided cyclophosphamide-induced ovarian insufficiency by remodeling the IGF-1-kisspeptin system, ovarian apoptosis, and angiogenesis.更正：表达Neat-1、Hotair-1、miR-21、miR-644和miR-144的骨髓间充质干细胞通过重塑IGF-1-促性腺激素释放激素神经元系统、卵巢细胞凋亡和血管生成来缓解环磷酰胺诱导的卵巢功能不全。
J Ovarian Res. 2024 Nov 29;17(1):239. doi: 10.1186/s13048-024-01564-4.
2
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本文引用的文献
1
Bone marrow mesenchymal stem cells expressing Neat-1, Hotair-1, miR-21, miR-644, and miR-144 subsided cyclophosphamide-induced ovarian insufficiency by remodeling the IGF-1-kisspeptin system, ovarian apoptosis, and angiogenesis.表达 Neat-1、Hotair-1、miR-21、miR-644 和 miR-144 的骨髓间充质干细胞通过重塑 IGF-1- kisspeptin 系统、卵巢细胞凋亡和血管生成来缓解环磷酰胺诱导的卵巢功能不全。
J Ovarian Res. 2024 Sep 12;17(1):184. doi: 10.1186/s13048-024-01498-x.
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