Correction: TSG101 depletion dysregulates mitochondria and PML NBs, triggering MAD2-overexpressing interphase cell death (MOID) through AIFM1-PML-DAXX pathway.
作者信息
Xi Yao, Xu Rui, Chen Shengnan, Fang Jiezhu, Duan Xiang, Zhang Yidan, Zhong Guoli, He Zhifei, Guo Yan, Li Xinyu, Tao Wenzhi, Li Yang, Li Yan, Fang Lei, Niikura Yohei
机构信息
National Resource Center for Mutant Mice, MOE Key Laboratory of Model Animals for Disease Study, Model Animal Research Center, Medical School of Nanjing University, Nanjing, 210061, China.
Jiangsu Key Laboratory of Molecular Medicine, Medical School of Nanjing University, Nanjing, 210032, China.
出版信息
Cell Death Dis. 2024 Dec 16;15(12):888. doi: 10.1038/s41419-024-07294-1.
Abstract
摘要
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Correction: TSG101 depletion dysregulates mitochondria and PML NBs, triggering MAD2-overexpressing interphase cell death (MOID) through AIFM1-PML-DAXX pathway.更正：TSG101缺失会使线粒体和早幼粒细胞白血病核体失调，通过AIFM1-PML-DAXX途径引发过表达MAD2的间期细胞死亡（MOID）。
Cell Death Dis. 2024 Dec 16;15(12):888. doi: 10.1038/s41419-024-07294-1.
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TSG101 depletion dysregulates mitochondria and PML NBs, triggering MAD2-overexpressing interphase cell death (MOID) through AIFM1-PML-DAXX pathway.TSG101 缺失会扰乱线粒体和 PML NBs，通过 AIFM1-PML-DAXX 途径触发 MAD2 过表达的有丝分裂期细胞死亡（MOID）。
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