Shiraishi Jun, Mabuchi Takashi, Kajihara Takashi, Ukawa Rikuya, Nishimura Tetsuro, Ohkura Takashi, Taminishi Shunta, Tsuji Yumika, Saburi Makoto, Takigami Masao, Tsubakimoto Yoshinori, Inoue Keiji, Ishibashi Kazuya
Department of Cardiology, Kyoto Second Red Cross Hospital, Kyoto, Japan.
Department of Second Laboratory Medicine, Kyoto Second Red Cross Hospital, Kyoto, Japan.
J Cardiol Cases. 2024 Aug 29;30(6):196-200. doi: 10.1016/j.jccase.2024.08.005. eCollection 2024 Dec.
Intervention to proximal lesions should be avoided in graft-protected native coronary arteries in general, because there might be a risk for bypass-graft failure. An 81-year-old man with coronary artery bypass grafting surgery due to 3-vessel disease 17 years previously complained of worsening angina. Coronary angiography (CAG) revealed a diseased saphenous vein graft (SVG) and a probable functional occlusion in the mid left anterior descending coronary artery (LAD) concomitant with calcified severe stenosis in the left main (LM)-proximal LAD, and patent right internal thoracic artery (RITA)-LAD graft. After the first percutaneous coronary intervention (PCI) against the SVG lesion, we performed second PCI against the LM-proximal LAD lesions to release angina symptom and prevent LM occlusion. After rotational atherectomy (RA) with 1.5/1.75 mm burrs and balloon dilations, we detected a slight antegrade flow to distal LAD. To preclude possibility of graft failure in the RITA, we did not add further large-balloon dilations and stent implantations, and finally dilated with 3.0-mm drug-coated balloons (DCBs), leading to angina-free condition. Six-month follow-up CAG revealed no further vessel narrowing in both target vessels without RITA-graft failure. Stent-less PCI using relatively small-sized RA/DCB might be feasible for native proximal calcified lesions with patent bypass graft.
•Full expansion of native proximal lesions should be avoided in internal thoracic artery (ITA) - protected coronary arteries in general, because it might provoke ITA-graft failure due to flow competition.•Stent-less modest dilation using relatively small-sized rotational atherectomy burr and drug-coated balloon might be a revascularization therapy of choice for native proximal calcified lesion with patent ITA bypass graft.
一般来说,应避免对移植血管保护的自身冠状动脉近端病变进行干预,因为可能存在旁路移植失败的风险。一名81岁男性,17年前因三支血管病变接受冠状动脉旁路移植手术,现诉心绞痛加重。冠状动脉造影(CAG)显示大隐静脉移植血管(SVG)病变,左前降支冠状动脉(LAD)中段可能存在功能性闭塞,同时左主干(LM)-LAD近端有钙化严重狭窄,右胸廓内动脉(RITA)-LAD移植血管通畅。在对SVG病变进行首次经皮冠状动脉介入治疗(PCI)后,我们对LM-LAD近端病变进行了第二次PCI,以缓解心绞痛症状并预防LM闭塞。在使用1.5/1.75毫米磨头进行旋磨术(RA)和球囊扩张后,我们检测到LAD远端有轻微的正向血流。为避免RITA移植血管失败的可能性,我们未进一步进行大球囊扩张和支架植入,最后用3.0毫米药物涂层球囊(DCB)进行扩张,使患者心绞痛症状消失。六个月的随访CAG显示,两个靶血管均未进一步狭窄,RITA移植血管未失败。对于有通畅旁路移植血管的自身近端钙化病变,使用相对小尺寸的RA/DCB进行无支架PCI可能是可行的。
•一般来说,应避免对胸廓内动脉(ITA)保护的冠状动脉自身近端病变进行充分扩张,因为这可能因血流竞争导致ITA移植血管失败。•使用相对小尺寸的旋磨术磨头和药物涂层球囊进行无支架适度扩张可能是有通畅ITA旁路移植血管的自身近端钙化病变的血管重建治疗选择。