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心肺复苏后假性降低血清钾水平。对评估急性心肌梗死中低钾血症的致心律失常性的意义。

Factitious lowering of the serum potassium level after cardiopulmonary resuscitation. Implications for evaluating the arrhythmogenicity of hypokalemia in acute myocardial infarction.

作者信息

Isner J M, Harten J T

出版信息

Arch Intern Med. 1985 Jan;145(1):161-2.

PMID:3970631
Abstract

Hypokalemia has been suggested as a predisposing factor to the development of fatal arrhythmias in acute myocardial infarction. Evidence cited to support this concept has been derived largely from studies in which the determination of the serum potassium level was made following a cardiac arrhythmia and/or arrest, and often following cardiopulmonary resuscitation (CPR); this postresuscitation potassium level has been considered to be representative of the prearrest value. In the patient described herein, serial determinations of serum potassium obtained fortuitously before and intentionally following sudden unexpected cardiac arrest in a hospitalized patient demonstrate that the prearrest serum potassium level cannot be inferred from electrolyte values obtained after CPR.

摘要

低钾血症被认为是急性心肌梗死发生致命性心律失常的一个易感因素。支持这一概念的证据主要来自于一些研究,在这些研究中,血清钾水平的测定是在心律失常和/或心脏骤停后,且常常是在心肺复苏(CPR)后进行的;复苏后的钾水平被认为代表了心脏骤停前的值。在本文所述的患者中,对一名住院患者在意外心脏骤停之前偶然获得以及之后有意测定的血清钾进行的系列测定表明,不能从心肺复苏后获得的电解质值推断心脏骤停前的血清钾水平。

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Arch Intern Med. 1985 Jan;145(1):161-2.
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