Levin V N, Murav'ev A V
Biull Eksp Biol Med. 1985 Feb;99(2):142-4.
It was established in chronic experiments on 26 dogs that long-term adaptation to regular muscular activity caused a 19.6-46.1% decrease in blood viscosity. Hematocrit was lowered and red cell deformability improved. A correlation was observed between blood viscosity and its oxygen transport function. As a result of muscular training the role of plasma protein in blood viscosity increased and that of red cells declined. Single muscular activity produced a 48.2-81% increase in blood viscosity, while plasma viscosity remained unchanged. Trained animals had lower absolute values of blood viscosity even at the time of muscular effort as compared to those in untrained animals at rest.
在对26只狗进行的慢性实验中发现，长期适应规律的肌肉活动会使血液粘度降低19.6%至46.1%。血细胞比容降低，红细胞变形能力提高。观察到血液粘度与其氧运输功能之间存在相关性。肌肉训练的结果是，血浆蛋白在血液粘度中的作用增加，而红细胞的作用下降。单次肌肉活动会使血液粘度增加48.2%至81%，而血浆粘度保持不变。与未训练的动物在休息时相比，训练有素的动物即使在进行肌肉运动时血液粘度的绝对值也较低。
