Jeewooth Avinash, Kaushik Atul, Jaswal Aparna, Kumar Vikas
Dept. of Electrophysiology, Fortis Escorts Heart Institute(FEHI), New Delhi, India.
Department of Cardiology, FEHI, Okhla, New Delhi, India.
Indian Pacing Electrophysiol J. 2025 Jan-Feb;25(1):50-53. doi: 10.1016/j.ipej.2024.12.004. Epub 2024 Dec 24.
The SA node is protected from the overdrive suppression by the other tissues or tachyarrhythmias by means of atrio-sinus entrance block. A 57-year-old woman presented with symptomatic tachy-brady syndrome and severe LV dysfunction. Electrophysiological study revealed presence of left lateral accessory pathway. After ablation of the accessory pathway, tachycardia could no longer be induced. Her LV systolic function significantly improved on follow up and an external loop recorder showed no episodes of bradycardia. Therefore, we conclude that the bradycardia was due to overdrive suppression of SA node caused by AVRT, and the reversible cardiomyopathy was also due to high tachycardia burden.
窦房结通过窦房入口阻滞,免受其他组织或快速性心律失常的超速抑制。一名57岁女性因症状性心动过缓-心动过速综合征和严重左心室功能障碍就诊。电生理研究显示存在左侧旁路。消融旁路后,不再能诱发心动过速。随访时其左心室收缩功能显著改善,体外环路记录仪未记录到心动过缓发作。因此,我们得出结论,心动过缓是由于房室折返性心动过速导致窦房结超速抑制,而可逆性心肌病也是由于高心动过速负荷所致。