Klein H, Schuilenburg R M, Janse M J, van Dam R T, Meijne N G, Rathsack P, Lichtlen P R
Z Kardiol. 1978 May;67(5):365-71.
This is a case report of a 60-year-old woman with a WPW-syndrome Type A developing persistent supraventricular tachycardia refractory to medical treatment. The re-entry mechanism was based on av-junction escape beats leading to retrograde conduction through the accessory pathway in presence of sinus node dysfunction i.e. long periods of sinus arrest probably enhanced through antiarrhythmic drug therapy and/or digitalis. With the aid of electrophysiological examinations and intraoperative epicardial mapping the accessory pathway was located in the upper lateral region of the left ventricle and interrupted by an incision in the av-groove from inside of the lfet atrium. Although an additional accessory pathway, not interrupted by surgery was suggested by the postoperative electrophysiological study, the patient remained free from tachycardia over more than 18 months, except for one episode terminated promptly by overdrive pacing from the right atrium. Postoperatively sinus bradycardia and av-junctional escape rhythm prevailed.
这是一例60岁患有A型预激综合征的女性病例报告,该患者出现了药物治疗无效的持续性室上性心动过速。折返机制基于房室交界区逸搏,在存在窦房结功能障碍时,即长时间的窦性停搏,可能通过抗心律失常药物治疗和/或洋地黄而加重,导致经旁路逆行传导。借助电生理检查和术中的心外膜标测,旁路位于左心室的上外侧区域,并通过从左心房内部在房室沟处的切口将其切断。尽管术后电生理研究提示存在一条未被手术切断的额外旁路,但患者在超过18个月的时间里未再发生心动过速,仅有一次发作通过右心房超速起搏迅速终止。术后以窦性心动过缓和房室交界区逸搏心律为主。