Gonzalez de la Rosa M, Perez Barreto L, del Rosario Cedres D
Graefes Arch Clin Exp Ophthalmol. 1985;222(4-5):182-5. doi: 10.1007/BF02133670.
Prostaglandins exercise different actions on the structures of the eye; among these is ocular hypertension. The mechanism by which ocular hypertension is provoked seems to be the breaking of the hematoaqueous barrier at the level of the iris and the ciliary processes. By fluorophotometry we studied the dynamics of the aqueous humor after topical administration of prostaglandin E-2 in rats. We observed an important reduction in flow that was interpreted as secondary to the increase in output resistance.
前列腺素对眼部结构有不同作用;其中之一是导致眼压升高。引发眼压升高的机制似乎是虹膜和睫状突水平处血-房水屏障的破坏。我们通过荧光光度法研究了大鼠局部应用前列腺素E-2后房水的动态变化。我们观察到房水流量显著减少,这被解释为是由于输出阻力增加所致。