Sato H, Fujiwara S, Otabe K, Sato S
No Shinkei Geka. 1985 Jan;13(1):117-21.
A 70-year-old woman was admitted to our hospital with attack of SAH. On admission, she was semi-comatose with no other neurological deficit. Left carotid angiography revealed an aneurysm of the anterior communicating artery and a large abnormal vessel connecting the left external carotid artery and the left vertebral artery. This large anastomotic vessel was thought to be primitive proatlantal intersegmental artery (proatlantal artery I). Right carotid angiography revealed a coiling of the right internal carotid artery and hypoplasia of the right A1 portion. Left retrograde brachial angiography revealed aplasia of the left vertebral artery. After admission her consciousness gradually improved but 17 days after admission she died of rerupture of the aneurysm. Autopsied brain showed that the left vertebral artery, namely proatlantal artery, was almost as large as the basilar artery and its macroscopical appearance was similar to other arteries. It was also obvious that the right vertebral artery was hypoplastic. As the rate of combination of the persistent artery with the intracranial aneurysm is relatively high, the authors think that some congenital factor may affect the occurrence of intracranial aneurysms.
一名70岁女性因蛛网膜下腔出血发作入住我院。入院时,她处于半昏迷状态,无其他神经功能缺损。左侧颈动脉血管造影显示前交通动脉瘤以及一条连接左颈外动脉和左椎动脉的粗大异常血管。这条粗大的吻合血管被认为是原始的寰前节间动脉(寰前动脉I)。右侧颈动脉血管造影显示右颈内动脉迂曲以及右A1段发育不全。左侧逆行肱动脉血管造影显示左椎动脉发育不全。入院后她的意识逐渐好转,但入院17天后死于动脉瘤再破裂。尸检大脑显示,左椎动脉,即寰前动脉,几乎与基底动脉一样粗大,其宏观外观与其他动脉相似。右椎动脉发育不全也很明显。由于永存动脉与颅内动脉瘤的合并率相对较高,作者认为某些先天性因素可能影响颅内动脉瘤的发生。