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When prenatal infection meets postnatal hyperoxia: Better models for bronchopulmonary dysplasia and its therapeutic approaches.

作者信息

Dong Ying, Leidner Annika, Marega Manuela, Rivetti Stefano, Bellusci Saverio

机构信息

Department of General Pediatrics and Neonatology, Justus-Liebig-University, Giessen 35392, Germany.

Universities of Giessen and Marburg Lung Center, Cardio-Pulmonary Institute, Giessen 35392, Germany.

出版信息

Chin Med J Pulm Crit Care Med. 2024 Dec 13;2(4):289-292. doi: 10.1016/j.pccm.2024.11.002. eCollection 2024 Dec.

DOI:10.1016/j.pccm.2024.11.002
PMID:39834586
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC11742557/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f657/11742557/0c3b0bdbea29/gr1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f657/11742557/0c3b0bdbea29/gr1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/f657/11742557/0c3b0bdbea29/gr1.jpg

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本文引用的文献

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Perinatal origins of bronchopulmonary dysplasia-deciphering normal and impaired lung development cell by cell.支气管肺发育不良的围产期起源——逐细胞解析正常和受损的肺发育
Mol Cell Pediatr. 2023 Apr 18;10(1):4. doi: 10.1186/s40348-023-00158-2.
2
Insights into the Black Box of Intra-Amniotic Infection and Its Impact on the Premature Lung: From Clinical and Preclinical Perspectives.羊膜内感染的黑箱及其对早产儿肺的影响的研究进展：从临床和临床前角度。
Int J Mol Sci. 2022 Aug 29;23(17):9792. doi: 10.3390/ijms23179792.
3
FGF10 Triggers De Novo Alveologenesis in a Bronchopulmonary Dysplasia Model: Impact on Resident Mesenchymal Niche Cells.
FGF10 触发支气管肺发育不良模型中的新生肺泡发生：对固有间充质细胞龛的影响。
Stem Cells. 2022 Jun 22;40(6):605-617. doi: 10.1093/stmcls/sxac025.
4
Prenatal Maternal Lipopolysaccharide and Mild Newborn Hyperoxia Increase Intrapulmonary Airway but Not Vessel Reactivity in a Mouse Model.产前母体脂多糖和轻度新生儿高氧增加小鼠模型肺内气道而非血管反应性。
Children (Basel). 2021 Mar 5;8(3):195. doi: 10.3390/children8030195.
5
Effects of antenatal lipopolysaccharide and postnatal hyperoxia on airway reactivity and remodeling in a neonatal mouse model.产前脂多糖和产后高氧对新生小鼠模型气道反应性和重塑的影响。
Pediatr Res. 2016 Mar;79(3):391-400. doi: 10.1038/pr.2015.232. Epub 2015 Nov 5.
6
Trends in Care Practices, Morbidity, and Mortality of Extremely Preterm Neonates, 1993-2012.1993 - 2012年极早产儿的护理实践、发病率及死亡率趋势
JAMA. 2015 Sep 8;314(10):1039-51. doi: 10.1001/jama.2015.10244.
7
Animal models of bronchopulmonary dysplasia. The term mouse models.支气管肺发育不良的动物模型。术语“鼠模型”。
Am J Physiol Lung Cell Mol Physiol. 2014 Dec 15;307(12):L936-47. doi: 10.1152/ajplung.00159.2014. Epub 2014 Oct 10.
8
Interleukin-1 receptor antagonist prevents murine bronchopulmonary dysplasia induced by perinatal inflammation and hyperoxia.白细胞介素-1 受体拮抗剂可预防围生期炎症和高氧诱导的小鼠支气管肺发育不良。
Proc Natl Acad Sci U S A. 2013 Aug 27;110(35):14384-9. doi: 10.1073/pnas.1306859110. Epub 2013 Aug 14.
9
Prenatal inflammation exacerbates hyperoxia-induced functional and structural changes in adult mice.产前炎症加重了高氧诱导的成年小鼠的功能和结构变化。
Am J Physiol Regul Integr Comp Physiol. 2012 Aug 1;303(3):R279-90. doi: 10.1152/ajpregu.00029.2012. Epub 2012 Jun 20.
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