Bardakhch'iah E A, Kirichenko Iu G
Tsitol Genet. 1985 Jan-Feb;19(1):3-5.
Ultrastructural alterations of adrenocorticocytes are sharply pronounced at the intermediate stage of endotoxin shock. The retrograde embolia by the chromaffine tissue elements of cortical vessels is a qualitatively new feature of the injury, which results in disadaptation and necrosis of adrenocorticocytes.
在内毒素休克的中期,肾上腺皮质细胞的超微结构改变极为明显。皮质血管嗜铬组织成分的逆行性栓子是损伤的一个全新质性特征,这会导致肾上腺皮质细胞功能失调和坏死。