Meerson F Z, Dosmagambetova R S
Kardiologiia. 1985 Apr;25(4):91-4.
A study of an isolated auricle showed that one day after the infarction the extensibility of the atrial myocardium was reduced, the Starling curve was depressed to result in about a two-fold decrease of the peak systolic tension, and atrial myocardial resistance to hypoxia and excessive calcium was also decreased. This complex of shifts was considerably diminished or eliminated altogether by means of building up an excessive glucose concentration or the administration of uridine, a cofactor of glycogen resynthesis. Therefore, the data obtained suggest that uridine can be used for correcting contractile disturbances of nonischemic compartments of the heart in myocardial infarction.
一项对离体耳廓的研究表明，梗死一天后心房心肌的伸展性降低，斯塔林曲线下移，导致收缩期峰值张力下降约两倍，并且心房心肌对缺氧和过量钙的耐受性也降低。通过提高葡萄糖浓度或给予尿苷（糖原再合成的辅助因子），这种复杂的变化显著减轻或完全消除。因此，所获得的数据表明，尿苷可用于纠正心肌梗死时心脏非缺血区的收缩功能紊乱。
