Beck Thomas R
Federal Office for Radiation Protection, Division of Environmental Radioactivity, Koepenicker Allee 120-130, 10318 Berlin, Germany.
J Radiol Prot. 2025 May 23;45(2). doi: 10.1088/1361-6498/add89d.
The study compares exposures to the lung caused by inhalation of radon and radon progeny with lung doses from external low-linear-energy-transfer (low-LET) radiation. For this purpose, lung cancer risks, lifetime effects and radiation detriments from chronic exposure to radon in homes and workplaces are calculated. The calculations apply accepted risk models derived from studies on miners and residential radon to the representative populations of International Commission on Radiological Protection (ICRP). In addition, comparable calculations are performed to determine risk quantities for the lung associated with low-LET radiation. In a working age population, a constant exposure to radon progeny of 1 working level month (WLM)/year results in a radiation detriment of 6.4∙10per year (1.8∙10yrat a constant exposure of 1 mJ·h·myr). For a whole population that is constantly exposed to a radon concentration of 100 Bq∙m, the radiation detriment is 1.1∙10per year. These values are based on epidemiological comparison using miner and residential studies separately. The relative biological effectiveness for alpha particles from the inhalation of radon and radon progeny is estimated to be around 10, which, despite of uncertainties, is significantly below the value of 20 recommended by ICRP for the radiation weighting factor of alpha particles. Comparison of results from epidemiological studies on radon in mines and in homes does not provide sufficient evidence that the fraction of unattached radon progeny has a significantly increased influence on the radiation risk und thus on the effective dose. An average annual effective dose of 6 mSv is determined for constant occupational exposure of a working age population to radon progeny with a rate of 1 WLM per year (1.7 mSv yrper mJ∙h∙myr). In the case of residential exposure, the average annual effective dose is 1.1 mSv, assuming that a whole population is constantly exposed to a radon activity concentration of 100 Bq∙m. The dose coefficients determined in this study are lower than the corresponding values derived from biokinetic and dosimetric models and currently recommended by ICRP.
该研究将吸入氡及其子体对肺部造成的照射与外部低线性能量转移(低LET)辐射产生的肺部剂量进行了比较。为此,计算了家庭和工作场所长期接触氡导致的肺癌风险、终生影响和辐射危害。这些计算将源自矿工和住宅氡研究的公认风险模型应用于国际放射防护委员会(ICRP)的代表性人群。此外,还进行了可比计算,以确定与低LET辐射相关的肺部风险量。在工作年龄人群中,每年持续接触1个工作水平月(WLM)的氡子体导致的辐射危害为每年6.4×10(每年持续接触1 mJ·h·m时为1.8×10)。对于持续暴露于氡浓度为100 Bq·m的整个人口,辐射危害为每年1.1×10。这些值分别基于使用矿工和住宅研究的流行病学比较。吸入氡及其子体产生的α粒子的相对生物效能估计约为10,尽管存在不确定性,但仍显著低于ICRP推荐的α粒子辐射权重因子值20。对矿井和家庭中氡的流行病学研究结果比较并未提供充分证据表明未附着的氡子体部分对辐射风险进而对有效剂量有显著增加的影响。对于工作年龄人群每年以1 WLM的速率持续职业性接触氡子体,确定的平均年有效剂量为6 mSv(每mJ∙h∙m每年为1.7 mSv)。在住宅暴露的情况下,假设整个人口持续暴露于100 Bq·m的氡活度浓度,平均年有效剂量为1.1 mSv。本研究确定的剂量系数低于ICRP目前推荐的源自生物动力学和剂量学模型的相应值。