Karsanov N V, Khananashvili M M, Nakhapetova Zh A, Suknidze Ts G, Kartvelishvili R G
Fiziol Zh SSSR Im I M Sechenova. 1985 Jan;71(1):113-9.
Experiments in dogs revealed that in informational neurosis the ability of myocardial contractile protein system (myocardial glycerinized filament bundles) to develop tension and produce work as well as the power of the contractile process are considerably reduced. Obviously, the energy production is not disturbed as a lower myocardial content of ATP (by 12%) and particularly of CP (by 54%) is caused by 8-9-month hypokinesia. Analogous shifts seem to occur in the human heart in informational neurosis which, in combination with the reduced intensity of the calcium transport by sarcoplasmic reticulum and mitochondria, may underlie the development of heart weakness.
对狗的实验表明，在信息性神经症中，心肌收缩蛋白系统（心肌甘油化细丝束）产生张力和做功的能力以及收缩过程的功率均显著降低。显然，能量产生并未受到干扰，因为8 - 9个月的运动减少导致心肌中ATP含量降低了12%，尤其是CP含量降低了54%。在人类心脏的信息性神经症中似乎也会出现类似的变化，这与肌浆网和线粒体钙转运强度降低相结合，可能是心脏衰弱发展的基础。
