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Letter to the Editor: "VEP Latency Delay Reflects Demyelination Beyond the Optic Nerve in the Cuprizone Model".

作者信息

Rocha Mendonça Henrique, de Pádua Ana Carolina, Augusto Alves da Cruz Saulo, Nascimento Pires Greice, Espírito-Santo Sheila

机构信息

Instituto de Biodiversidade e Sustentabilidade NUPEM, Universidade Federal do Rio de Janeiro, Macaé, Brazil.

Laboratório de Neurodegeneração e Reparo, Departamento de Patologia, Faculdade de Medicina, Universidade Federal do Rio de Janeiro, Rio de Janeiro, Brazil.

出版信息

Invest Ophthalmol Vis Sci. 2025 Jun 2;66(6):27. doi: 10.1167/iovs.66.6.27.

DOI:10.1167/iovs.66.6.27
PMID:40488715
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC12161388/
Abstract
摘要

相似文献

1
Letter to the Editor: "VEP Latency Delay Reflects Demyelination Beyond the Optic Nerve in the Cuprizone Model".致编辑的信:“在铜螯合剂模型中,视觉诱发电位潜伏期延迟反映了视神经以外的脱髓鞘病变”
Invest Ophthalmol Vis Sci. 2025 Jun 2;66(6):27. doi: 10.1167/iovs.66.6.27.
2
VEP Latency Delay Reflects Demyelination Beyond the Optic Nerve in the Cuprizone Model.VEP 潜伏期延迟反映了 Cuprizone 模型中视神经以外的脱髓鞘。
Invest Ophthalmol Vis Sci. 2024 Nov 4;65(13):50. doi: 10.1167/iovs.65.13.50.
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Latency delay of visual evoked potential is a real measurement of demyelination in a rat model of optic neuritis.视觉诱发电位潜伏期延迟是视神经炎大鼠模型脱髓鞘的真实测量。
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Transcranial direct current stimulation as a potential remyelinating therapy: Visual evoked potentials recovery in cuprizone demyelination.经颅直流电刺激作为一种潜在的髓鞘修复治疗方法:在脱髓鞘的铜中毒模型中视觉诱发电位的恢复。
Exp Neurol. 2024 Dec;382:114972. doi: 10.1016/j.expneurol.2024.114972. Epub 2024 Sep 24.
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Inflammatory demyelination alters subcortical visual circuits.炎症性脱髓鞘改变皮质下视觉回路。
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Experimental demyelination caused by primary oligodendrocyte dystrophy. Regional distribution of the lesions in the nervous system of mice [corrected].原发性少突胶质细胞营养不良引起的实验性脱髓鞘。小鼠神经系统病变的区域分布[已修正]
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Visual Evoked Potentials to Monitor Myelin Cuprizone-Induced Functional Changes.视觉诱发电位监测髓鞘铜螯合物诱导的功能变化。
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Brain. 1987 Feb;110 ( Pt 1):107-20. doi: 10.1093/brain/110.1.107.

本文引用的文献

1
The protective effect of Fingolimod upon visual behavior in a demyelination animal model is associated with synaptopathy prevention.芬戈莫德在脱髓鞘动物模型中对视觉行为的保护作用与预防突触病变有关。
Neurotoxicology. 2025 May;108:113-122. doi: 10.1016/j.neuro.2025.03.004. Epub 2025 Mar 12.
2
Therapeutic potential of pranlukast against cuprizone-induced inflammatory demyelination and sensory impairment in mice: Comparison with fingolimod.普仑司特对小鼠 cuprizone 诱导的炎性脱髓鞘和感觉障碍的治疗潜力:与芬戈莫德的比较。
Neurotoxicology. 2025 Mar;107:37-52. doi: 10.1016/j.neuro.2025.01.004. Epub 2025 Jan 31.
3
VEP Latency Delay Reflects Demyelination Beyond the Optic Nerve in the Cuprizone Model.VEP 潜伏期延迟反映了 Cuprizone 模型中视神经以外的脱髓鞘。
Invest Ophthalmol Vis Sci. 2024 Nov 4;65(13):50. doi: 10.1167/iovs.65.13.50.
4
Inflammatory demyelination alters subcortical visual circuits.炎症性脱髓鞘改变皮质下视觉回路。
J Neuroinflammation. 2017 Aug 18;14(1):162. doi: 10.1186/s12974-017-0936-0.
5
Synaptopathy connects inflammation and neurodegeneration in multiple sclerosis.突触病将多发性硬化症中的炎症和神经退行性变联系起来。
Nat Rev Neurol. 2015 Dec;11(12):711-24. doi: 10.1038/nrneurol.2015.222. Epub 2015 Nov 20.
6
Bicuculline enhances a negative component and diminishes a positive component of the visual evoked cortical potential in the cat.荷包牡丹碱增强猫视觉诱发皮层电位的负向成分并减弱其正向成分。
Proc Natl Acad Sci U S A. 1980 Dec;77(12):7476-8. doi: 10.1073/pnas.77.12.7476.