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Expression of concern: "RvD1binding with FPR2 attenuates inflammation via Rac1/NOX2 pathway after neonatal hypoxic-ischemic injury in rats" [EXP NEUROL, Volume 320 (2019) 112982].

出版信息

Exp Neurol. 2025 Sep;391:115293. doi: 10.1016/j.expneurol.2025.115293. Epub 2025 May 13.

DOI:10.1016/j.expneurol.2025.115293
PMID:40499995
Abstract
摘要

相似文献

1
Expression of concern: "RvD1binding with FPR2 attenuates inflammation via Rac1/NOX2 pathway after neonatal hypoxic-ischemic injury in rats" [EXP NEUROL, Volume 320 (2019) 112982].
Exp Neurol. 2025 Sep;391:115293. doi: 10.1016/j.expneurol.2025.115293. Epub 2025 May 13.
2
RvD1binding with FPR2 attenuates inflammation via Rac1/NOX2 pathway after neonatal hypoxic-ischemic injury in rats.RvD1 与 FPR2 结合通过 Rac1/NOX2 通路减轻大鼠新生缺氧缺血性损伤后的炎症反应。
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Curcumin attenuates hypoxic-ischemic brain injury in neonatal rats through induction of nuclear factor erythroid-2-related factor 2 and heme oxygenase-1.姜黄素通过诱导核因子红细胞2相关因子2和血红素加氧酶-1减轻新生大鼠缺氧缺血性脑损伤。
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Activation of MC1R with BMS-470539 attenuates neuroinflammation via cAMP/PKA/Nurr1 pathway after neonatal hypoxic-ischemic brain injury in rats.BMS-470539 通过 cAMP/PKA/Nurr1 通路激活 MC1R，减轻大鼠新生缺氧缺血性脑损伤后的神经炎症。
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Oxid Med Cell Longev. 2021 Feb 12;2021:6643171. doi: 10.1155/2021/6643171. eCollection 2021.