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阵发性室上性心动过速的电生理研究

[Electrophysiologic study of paroxysmal supraventricular tachycardia].

作者信息

Sakurai M

出版信息

Hokkaido Igaku Zasshi. 1985 Jul;60(4):487-98.

PMID:4054819
Abstract

Seventy patients (pts) with paroxysmal supraventricular tachycardia (PSVT) were examined by using electrophysiological techniques (EPS). At rest, 23 pts had the ECG evidence of W-P-W syndrome (group I) and 47 pts did not have (group II). In 20 pts of group I, PSVT could be induced by EPS. In 19 pts of them, the mechanism of PSVT was reentry using the accessory pathway (AP) retrogradely and the normal pathway (NP) antegradely, but only one showed the antidromic type (the former in reverse) of PSVT. Whereas in 45 pts of group II, PSVT could be induced reproduceably by EPS. In 31 pts of them, the mechanism of PSVT was reentry using concealed AP retrogradely and NP antegradely. In 13 pts, the mechanism was reentry within the AV node because dual pathways were found. In 11 pts, the reentrant beats conducted on the slow pathway (SP) antegradely, and on the fast pathway (FP) retrogradely. In 2 pts, reentrant beats conducted on the FP antegradely and on SP retrogradely. In one pt, PSVT was caused by sinus nodal or intraatrial reentry. Among 13 pts, in whom PSVT was caused by reentry using AP accompanied with dual pathways, the sudden change of the cycle length of PSVT were observed in 5 pts. PSVT which could only be induced by EPS in the laboratory were as follows: Af in 5, VT in 3 and sinus nodal reentrant tachycardia in one. In conclusion, in 65 pts of 70 pts with PSVT (93%), PSVT could be induced by EPS reproduceably. On the basis of EPS, the mechanism of PSVT was reentry involving AP in 29%, reentry involving concealed AP in 44%, AV nodal reentry in 19% and sinus nodal reentry in 1%.

摘要

采用电生理技术(EPS)对70例阵发性室上性心动过速(PSVT)患者进行了检查。静息时,23例患者有预激综合征的心电图证据(I组),47例患者没有(II组)。I组的20例患者中,PSVT可由EPS诱发。其中19例患者,PSVT的机制是利用旁路(AP)逆向和正常通路(NP)顺向折返,但只有1例表现为逆向型(前者相反)PSVT。而II组的45例患者中,PSVT可由EPS重复诱发。其中31例患者,PSVT的机制是利用隐匿性AP逆向和NP顺向折返。13例患者,机制是房室结内折返,因为发现了双径路。11例患者,折返搏动沿慢径路(SP)顺向传导,沿快径路(FP)逆向传导。2例患者,折返搏动沿FP顺向传导,沿SP逆向传导。1例患者,PSVT由窦房结或心房内折返引起。在13例PSVT由AP伴双径路折返引起的患者中,5例观察到PSVT的周期长度突然改变。仅在实验室中由EPS诱发的PSVT如下:房颤5例,室性心动过速3例,窦房结折返性心动过速1例。总之,70例PSVT患者中的65例(93%),PSVT可由EPS重复诱发。基于EPS,PSVT的机制为涉及AP的折返占29%,涉及隐匿性AP的折返占44%,房室结折返占19%,窦房结折返占1%。

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