Vomberg H E, Skrandies W
Klin Monbl Augenheilkd. 1985 Sep;187(3):205-8. doi: 10.1055/s-2008-1051018.
Cortical potentials evoked by dynamic random dot stereograms were recorded in 15 normal subjects and 19 patients with impaired stereoscopic vision. The potentials obtained were compaired with those evoked by similarly arranged checkerboard contrast stimuli, binocularly presented. The two modes of stimulation yielded potentials of similar component latencies but the stereoscopically evoked component amplitudes were significantly lower. In nine-channel recordings the stereoscopically evoked potentials were found to be located farther anterior topographically, suggesting a different neural generating mechanism. Patients with impaired stereoscopic vision had lower amplitudes and longer peak latencies than normal subjects. In addition, larger disparities had to be used to elicit a response in these patients. There was a high degree of correlation between the disparity thresholds determined by evoked cortical potentials and those obtained by sensory measurements.
在15名正常受试者和19名立体视觉受损患者中记录了动态随机点立体图诱发的皮质电位。将获得的电位与双眼呈现的、排列相似的棋盘格对比刺激诱发的电位进行比较。两种刺激模式产生的电位成分潜伏期相似，但立体视觉诱发的成分振幅明显较低。在九通道记录中，发现立体视觉诱发的电位在地形上位于更靠前的位置，这表明存在不同的神经产生机制。立体视觉受损的患者比正常受试者的振幅更低，峰值潜伏期更长。此外，在这些患者中必须使用更大的视差来引发反应。通过诱发皮质电位确定的视差阈值与通过感觉测量获得的视差阈值之间存在高度相关性。
