更正：在高脂饮食诱导的非酒精性脂肪性肝炎小鼠模型中，脂肪变性参与了肾脏疾病的进展。

Correction: Steatosis is involved in the progression of kidney disease in a high-fat-diet-induced non-alcoholic steatohepatitis mouse model.

出版信息

PLoS One. 2025 Aug 4;20(8):e0329654. doi: 10.1371/journal.pone.0329654. eCollection 2025.

DOI:10.1371/journal.pone.0329654

PMID:40758620

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC12321048/

Abstract

[This corrects the article DOI: 10.1371/journal.pone.0265461.].

摘要

[本文更正了文章的数字对象标识符：10.1371/journal.pone.0265461。]

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c86e/12321048/b3b502836692/pone.0329654.g002.jpg

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Correction: Steatosis is involved in the progression of kidney disease in a high-fat-diet-induced non-alcoholic steatohepatitis mouse model.更正：在高脂饮食诱导的非酒精性脂肪性肝炎小鼠模型中，脂肪变性参与了肾脏疾病的进展。

PLoS One. 2025 Aug 4;20(8):e0329654. doi: 10.1371/journal.pone.0329654. eCollection 2025.

Steatosis is involved in the progression of kidney disease in a high-fat-diet-induced non-alcoholic steatohepatitis mouse model.脂肪变性参与高脂肪饮食诱导的非酒精性脂肪性肝炎小鼠模型中肾脏疾病的进展。

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Elevated Kallistatin promotes the occurrence and progression of non-alcoholic fatty liver disease.升高的 Kallistatin 促进非酒精性脂肪性肝病的发生和进展。

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PLoS One. 2022 Mar 16;17(3):e0265461. doi: 10.1371/journal.pone.0265461. eCollection 2022.

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更正：在高脂饮食诱导的非酒精性脂肪性肝炎小鼠模型中，脂肪变性参与了肾脏疾病的进展。

Correction: Steatosis is involved in the progression of kidney disease in a high-fat-diet-induced non-alcoholic steatohepatitis mouse model.

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