Patrick J, Golden M
Am J Clin Nutr. 1977 Sep;30(9):1478-81. doi: 10.1093/ajcn/30.9.1478.
Intracellular electrolytes and sodium transport were measured in leukocytes obtained from malnourished children. In the presence of edema, leukocyte sodium and potassium were raised. The total flux and the glycoside-sensitive portion were increased. Loss of edema was associated with reductions in all these measurements. In marasmus, glycoside-sensitive sodium efflux was reduced compared to recovered values. Sodium was increased and potassium reduced. It is concluded that at least two defects in sodium transport may occur in protein energy malnutrition, an increased passive permeability in kwashiorkor and a reduced active transport for sodium in marasmus.
对营养不良儿童的白细胞进行了细胞内电解质和钠转运的测量。在出现水肿的情况下，白细胞中的钠和钾含量升高。总通量和对糖苷敏感的部分增加。水肿的消退与所有这些测量值的降低有关。在消瘦症中，与恢复值相比，对糖苷敏感的钠外流减少。钠增加而钾减少。得出的结论是，蛋白质能量营养不良可能至少出现两种钠转运缺陷，即夸希奥科病中被动通透性增加以及消瘦症中钠的主动转运减少。
