Fernandes Steve, Jeevannavar Santosh, Shenoy Keshav, Baindoor Prasanna
Department of Orthopaedics, SDM Medical College and Hospital, Dharwad, Karnataka, India.
J Orthop Case Rep. 2025 Sep;15(9):116-120. doi: 10.13107/jocr.2025.v15.i09.6036.
The acromioclavicular (AC) ganglion cyst first described by a shoulder arthrogram by Craig in 1984, still remains an uncommon presentation of a shoulder pathology. Classified as a type one cyst by Hiller in the presence of a rotator cuff pathology, the management of these lesions is governed by ill-defined guidelines. Literature shows the variability of surgical management ranges from simple cyst excision and lateral end clavicular excision to reverse shoulder arthroplasty. Type two cysts are usually benign ganglion cysts, filled with mucinous putty material, and arise from the AC joint as a result of constant pressure of the synovial fluid passing in sequence, from the glenohumeral joint to the subacromial bursa and then reaching the AC joint. They are painless, gradually enlarging masses that appear just at the tip of the shoulder and, unless large, do not limit shoulder movements.
We present to you a 77-year-old male patient with hypertension and diabetes, who came with a painless swelling over the right shoulder, gradually increasing in size for the first 6 months. He gave a prior history of pain in the shoulder associated with stiffness, depicting a frozen shoulder 5 years ago. On examination, the swelling was soft, cystic, non-reducible, fluctuant, measuring 3 × 3 cm centred over the AC joint. The range of motion was near normal, comparable to the opposite side, associated with crepitus, although pain free. Clinical special tests revealed intact but weak cuff muscles. Imaging was performed that revealed superior migration of the humeral head and near complete chronic supraspinatus tear with glenohumeral arthritis on radiograph. Magnetic resonance imaging showed synovial thickening with a cystic homogenous swelling above the AC joint, a cutoff geyser sign with no communication to the AC joint or subacromial bursa. Complete excision of the cyst in toto was performed with no additional procedure. The histopathological examination revealed a ganglion cyst.
The AC joint cyst is a rare clinical diagnosis, requiring further insight into the spectrum of management of these lesions. In our case, the lack of clinical findings makes the management more challenging and thus, individualised.
肩锁关节(AC)腱鞘囊肿于1984年由克雷格通过肩关节造影首次描述,至今仍是一种不常见的肩部病变表现。希勒在存在肩袖病变的情况下将其分类为I型囊肿,这些病变的处理遵循不明确的指南。文献表明,手术治疗方法的差异很大,从简单的囊肿切除和锁骨外侧端切除到反式肩关节置换术。II型囊肿通常是良性腱鞘囊肿,充满黏液样物质,由于滑液从盂肱关节依次流经肩峰下囊然后到达肩锁关节的持续压力而起源于肩锁关节。它们是无痛、逐渐增大的肿块,出现在肩部顶端,除非体积较大,一般不限制肩部活动。
我们向您介绍一位77岁的男性患者,患有高血压和糖尿病,因右肩部无痛性肿胀前来就诊,最初6个月肿胀逐渐增大。他有5年前肩部疼痛伴僵硬的病史,当时诊断为肩周炎。检查时,肿胀柔软、呈囊性、不可复位、有波动感,位于肩锁关节上方,大小为3×3厘米。活动范围接近正常,与对侧相当,伴有摩擦音,但无疼痛。临床特殊检查显示肩袖肌肉完整但力量减弱。影像学检查显示X线片上肱骨头向上移位,肩袖近完全慢性撕裂伴盂肱关节炎。磁共振成像显示滑膜增厚,肩锁关节上方有囊性均匀肿胀,有截断性喷泉征,与肩锁关节或肩峰下囊无相通。完整切除整个囊肿,未进行其他手术。组织病理学检查显示为腱鞘囊肿。
肩锁关节囊肿是一种罕见的临床诊断,需要进一步深入了解这些病变的处理范围。在我们的病例中,缺乏临床表现使处理更具挑战性,因此需要个体化治疗。