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Kinetic and thermodynamic aspects of the mechanism of general anesthesia in a model system of firefly luminescence in vitro.

作者信息

Ueda I, Kamaya H

出版信息

Anesthesiology. 1973 May;38(5):425-36. doi: 10.1097/00000542-197305000-00002.

DOI:10.1097/00000542-197305000-00002
PMID:4145377
Abstract
摘要

相似文献

1
Kinetic and thermodynamic aspects of the mechanism of general anesthesia in a model system of firefly luminescence in vitro.体外萤火虫发光模型系统中全身麻醉机制的动力学和热力学方面
Anesthesiology. 1973 May;38(5):425-36. doi: 10.1097/00000542-197305000-00002.
2
Kinetics of the firefly luciferase catalyzed reactions.萤火虫荧光素酶催化反应的动力学
Biochemistry. 1974 Feb 26;13(5):921-5. doi: 10.1021/bi00702a015.
3
Inhibition of a brain protease by general anaesthetics.
Nature. 1965 Jul 24;207(995):419-20. doi: 10.1038/207419a0.
4
A quantitative slide test for ATP.
Experientia. 1970;26(2):221-2. doi: 10.1007/BF01895599.
5
Anesthetic inhibition of firefly luciferase, a protein model for general anesthesia, does not exhibit pressure reversal.麻醉剂对萤火虫荧光素酶（一种全身麻醉的蛋白质模型）的抑制作用不表现出压力逆转。
Biophys J. 1991 Dec;60(6):1309-14. doi: 10.1016/S0006-3495(91)82168-3.
6
Does pressure antagonize anesthesia? Opposite effects on specific and nonspecific inhibitors of firefly luciferase.压力会拮抗麻醉作用吗？对萤火虫荧光素酶特异性和非特异性抑制剂的相反作用。
Biophys J. 1999 Jan;76(1 Pt 1):483-8. doi: 10.1016/S0006-3495(99)77216-4.
7
Molecular mechanism of inhibition of firefly luminescence by local anesthetics.局部麻醉药抑制萤火虫发光的分子机制。
Proc Natl Acad Sci U S A. 1976 Feb;73(2):481-5. doi: 10.1073/pnas.73.2.481.
8
An investigation of myoplasmic magnesium adenosine triphosphate in barnacle muscle fibres with the firefly method.用萤火虫法对藤壶肌纤维中的肌质镁三磷酸腺苷进行的研究。
J Physiol. 1980 May;302:73-88. doi: 10.1113/jphysiol.1980.sp013230.
9
Effect of temperature on potency of anesthetic agents.温度对麻醉剂效能的影响。
J Appl Physiol. 1974 Oct;37(4):552-5. doi: 10.1152/jappl.1974.37.4.552.
10
[Application of the theory of absolute reaction rate to the mechanism of general anesthesia--hydrophobic inhibition of firefly luminescence by anesthetics].[绝对反应速率理论在全身麻醉机制中的应用——麻醉剂对萤火虫发光的疏水抑制作用]
Masui. 1973 Jul;22(7):626-37.

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1
Clinical concentrations of chemically diverse general anesthetics minimally affect lipid bilayer properties.化学性质各异的全身麻醉药的临床浓度对脂质双层特性的影响极小。
Proc Natl Acad Sci U S A. 2017 Mar 21;114(12):3109-3114. doi: 10.1073/pnas.1611717114. Epub 2017 Mar 6.
2
Mechanistic Insights into the Modulation of Voltage-Gated Ion Channels by Inhalational Anesthetics.吸入性麻醉剂对电压门控离子通道调节作用的机制性见解
Biophys J. 2015 Nov 17;109(10):2003-11. doi: 10.1016/j.bpj.2015.09.032.
3
Volatile anesthetics inhibit sodium channels without altering bulk lipid bilayer properties.
挥发性麻醉剂抑制钠通道，而不改变脂质双分子层的整体特性。
J Gen Physiol. 2014 Dec;144(6):545-60. doi: 10.1085/jgp.201411172. Epub 2014 Nov 10.
4
The location and nature of general anesthetic binding sites on the active conformation of firefly luciferase; a time resolved photolabeling study.萤火虫荧光素酶活性构象上全麻结合位点的位置和性质；时间分辨光标记研究。
PLoS One. 2012;7(1):e29854. doi: 10.1371/journal.pone.0029854. Epub 2012 Jan 17.
5
Effect of inhalation anesthetics on swimming activity of artemia salina.吸入麻醉剂对卤虫游泳活动的影响。
J Anesth. 1991 Jul;5(3):287-93. doi: 10.1007/s0054010050287.
6
Does pressure antagonize anesthesia? Opposite effects on specific and nonspecific inhibitors of firefly luciferase.压力会拮抗麻醉作用吗？对萤火虫荧光素酶特异性和非特异性抑制剂的相反作用。
Biophys J. 1999 Jan;76(1 Pt 1):483-8. doi: 10.1016/S0006-3495(99)77216-4.
7
Structural basis for the inhibition of firefly luciferase by a general anesthetic.一种全身麻醉剂抑制萤火虫荧光素酶的结构基础。
Biophys J. 1998 Nov;75(5):2205-11. doi: 10.1016/S0006-3495(98)77664-7.
8
Is there a specific receptor for anesthetics? Contrary effects of alcohols and fatty acids on phase transition and bioluminescence of firefly luciferase.麻醉剂是否存在特定受体？醇类和脂肪酸对萤火虫荧光素酶相变和生物发光的相反作用。
Biophys J. 1998 Aug;75(2):1052-7. doi: 10.1016/S0006-3495(98)77594-0.
9
Thermodynamics of anesthetic/protein interactions. Temperature studies on firefly luciferase.麻醉剂/蛋白质相互作用的热力学。萤火虫荧光素酶的温度研究。
Biophys J. 1993 Apr;64(4):1264-71. doi: 10.1016/S0006-3495(93)81491-7.
10
Inhibition by anaesthetics of 14C-guanidinium flux through the voltage-gated sodium channel and the cation channel of the 5-HT3 receptor of N1E-115 neuroblastoma cells.麻醉剂对14C-胍盐通过N1E-115神经母细胞瘤细胞电压门控钠通道和5-HT3受体阳离子通道的通量的抑制作用。
Naunyn Schmiedebergs Arch Pharmacol. 1993 Feb;347(2):125-32. doi: 10.1007/BF00169256.