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Oxidative demethylation of 14C-griseofulvin by liver microsomes of rats and mice.

作者信息

Chang R L, Symchowicz S, Lin C C

出版信息

Biochem Pharmacol. 1973 Jun 1;22(11):1389-92. doi: 10.1016/0006-2952(73)90314-6.

DOI:10.1016/0006-2952(73)90314-6
PMID:4146984
Abstract
摘要

相似文献

1
Oxidative demethylation of 14C-griseofulvin by liver microsomes of rats and mice.大鼠和小鼠肝脏微粒体对14C-灰黄霉素的氧化脱甲基作用。
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引用本文的文献

1
Ferrochelatase and N-alkylated porphyrins.亚铁螯合酶与N-烷基化卟啉
Mol Cell Biochem. 1984 Sep;64(2):127-37. doi: 10.1007/BF00224769.
2
Isolation of two N-monosubstituted protoporphyrins, bearing either the whole drug or a methyl group on the pyrrole nitrogen atom, from liver of mice given griseofulvin.从小鼠肝脏中分离出两种 N-单取代原卟啉，它们在吡咯氮原子上分别带有整个药物或一个甲基，这些小鼠曾给予灰黄霉素。
Biochem J. 1991 Mar 15;274 ( Pt 3)(Pt 3):843-8. doi: 10.1042/bj2740843.
3
Stimulation of the pathway of porphyrin synthesis in the liver of rats and mice by griseofulvin, 3,5-Diethoxycarbonyl-1,4-dihydrocollidine and related drugs: evidence for two basically different mechanisms.
灰黄霉素、3,5-二乙氧羰基-1,4-二氢可力丁及相关药物对大鼠和小鼠肝脏卟啉合成途径的刺激作用：两种根本不同机制的证据
Biochem J. 1975 Jan;146(1):285-7. doi: 10.1042/bj1460285.