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J Clin Pathol. 1973 Sep;26(9):684-6. doi: 10.1136/jcp.26.9.684.
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RAPID ELECTROPHORETIC SEPARATION OF LACTATE DEHYDROGENASE ISOENZYMES ON CELLULOSE ACETATE.乳酸脱氢酶同工酶在醋酸纤维素上的快速电泳分离
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Serum gamma-glutamyl transpeptidase activity in myocardial infarction.心肌梗死时血清γ-谷氨酰转肽酶活性
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Gamma-glutamyl-transpeptidase in myocardial infarction. Clinical and experimental studies.心肌梗死中的γ-谷氨酰转肽酶。临床与实验研究。
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-glutamyl-transpeptidase in diseases of the liver, cardiovascular system and diabetes mellitus.肝脏、心血管系统疾病及糖尿病中的γ-谷氨酰转肽酶
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Serum peptidases in myocardial infarction.心肌梗死中的血清肽酶。
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心肌梗死后的血清D-谷氨酰转移酶

Serum D-glutamyl transferase following myocardial infarction.

作者信息

Connell M D

出版信息

J Clin Pathol. 1973 Sep;26(9):684-6. doi: 10.1136/jcp.26.9.684.

DOI:10.1136/jcp.26.9.684
PMID:4148076
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC477850/
Abstract

D-glutamyl transferase (GMT) activity was measured in 49 patients with proven myocardial infarction. Twenty-three patients had normal GMT activity and 26 had increased GMT activity. Most of the patients with increased GMT had evidence of liver dysfunction and it is suggested that any increases in serum GMT activity following myocardial infarction are a result of secondary liver damage rather than a release of GMT from cardiac tissue.

摘要

对49例经证实患有心肌梗死的患者测定了D - 谷氨酰转移酶(GMT)活性。23例患者的GMT活性正常,26例患者的GMT活性升高。大多数GMT活性升高的患者有肝功能障碍的证据,提示心肌梗死后血清GMT活性的任何升高都是继发性肝损伤的结果,而非来自心脏组织的GMT释放所致。