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Formation and binding of carbanions by cytochrome P-450 of liver microsomes.

作者信息

Ullrich V, Schnabel K H

出版信息

Drug Metab Dispos. 1973 Jan-Feb;1(1):176-83.

PMID:4149380
Abstract
摘要

相似文献

1
Formation and binding of carbanions by cytochrome P-450 of liver microsomes.
Drug Metab Dispos. 1973 Jan-Feb;1(1):176-83.
2
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引用本文的文献

1
Inhibition and induction of cytochrome P450 and the clinical implications.细胞色素P450的抑制与诱导及其临床意义。
Clin Pharmacokinet. 1998 Nov;35(5):361-90. doi: 10.2165/00003088-199835050-00003.
2
The mechanism of halothane binding to microsomal cytochrome P450.
Naunyn Schmiedebergs Arch Pharmacol. 1974;285(4):315-24. doi: 10.1007/BF00501461.
3
Metabolic activation of halothane and its covalent binding to liver endoplasmic proteins in vitro.氟烷的代谢活化及其在体外与肝脏内质网蛋白的共价结合。
Naunyn Schmiedebergs Arch Pharmacol. 1973;279(1):39-52. doi: 10.1007/BF00502066.
4
Molecular aspects of catechol and pyrogallol inhibition of liver microsomal lipid peroxidation stimulated by ferrous ion-ADP-complexes or by carbon tetrachloride.儿茶酚和连苯三酚对亚铁离子 - ADP - 复合物或四氯化碳刺激的肝微粒体脂质过氧化的抑制作用的分子机制
Naunyn Schmiedebergs Arch Pharmacol. 1977 Nov;300(2):179-87. doi: 10.1007/BF00505049.
5
A comparative study on the irreversible binding of labeled halothane trichlorofluoromethane, chloroform, and carbon tetrachloride to hepatic protein and lipids in vitro and in vivo.标记的氟烷、三氯氟甲烷、氯仿和四氯化碳在体外和体内与肝脏蛋白质和脂质不可逆结合的比较研究。
Arch Toxicol. 1975 Dec 18;34(4):289-308. doi: 10.1007/BF00353849.