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Demonstration of copper and acid phosphatase activity in hepatocyte lysosomes in experimental copper toxicity.

作者信息

Goldfischer S

出版信息

Nature. 1967 Jul 1;215(5096):74-5. doi: 10.1038/215074a0.

DOI:10.1038/215074a0
PMID:4167884
Abstract
摘要

相似文献

1
Demonstration of copper and acid phosphatase activity in hepatocyte lysosomes in experimental copper toxicity.实验性铜中毒时肝细胞溶酶体中铜及酸性磷酸酶活性的显示
Nature. 1967 Jul 1;215(5096):74-5. doi: 10.1038/215074a0.
2
Copper in hepatocyte lysosomes of the toad, Bufo marinus L.
Nature. 1970 Oct 10;228(5267):172-3. doi: 10.1038/228172a0.
3
Studies on the pathogenesis of hepatolenticular degeneration. 3. The effect of copper on rat liver lysosomes.肝豆状核变性发病机制的研究。3. 铜对大鼠肝脏溶酶体的影响。
Am J Pathol. 1968 Dec;53(6):903-27.
4
THE LOCALIZATION OF COPPER IN THE PERICANALICULAR GRANULES (LYSOSOMES) OF LIVER IN WILSON'S DISEASE (HEPATOLENTICULAR DEGENERATION) .威尔逊氏病(肝豆状核变性)中肝脏胆小管周围颗粒(溶酶体)内铜的定位
Am J Pathol. 1965 Jun;46(6):977-83.
5
Studies on the pathogenesis of hepatolenticular degeneration. I. Acid phosphatase activity in copper-loaded rat livers.肝豆状核变性发病机制的研究。I. 铜负荷大鼠肝脏中的酸性磷酸酶活性
Am J Pathol. 1967 Oct;51(4):471-81.
6
Electron probe microanalysis of liver in Wilson's disease. Simultaneous assay for copper and for lead deposited by acid phosphatase activity in lysosomes.肝豆状核变性患者肝脏的电子探针微量分析。溶酶体中酸性磷酸酶活性沉积的铜和铅的同步测定。
Am J Pathol. 1966 Feb;48(2):305-15.
7
Changes in the distribution of hepatic copper in relation to the progression of Wilson's disease (hepatolenticular degeneration).肝豆状核变性(威尔逊病)进展过程中肝脏铜分布的变化
Am J Pathol. 1968 Dec;53(6):883-901.
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Similarity of chronic copper toxicity in rats to copper deposition of Wilson's disease.大鼠慢性铜中毒与威尔逊病铜沉积的相似性。
Mayo Clin Proc. 1974 Jun;49(6):368-75.
9
Detection of distribution of copper inside and outside of lysosomes in cultured hepatolenticular degeneration fibroblasts by electron probe X-ray microanalysis.
Hepatobiliary Pancreat Dis Int. 2003 May;2(2):270-3.
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Lysosomal defect of hepatic copper excretion in Wilson's disease (hepatolenticular degeneration).威尔逊病(肝豆状核变性)中肝脏铜排泄的溶酶体缺陷。
Gastroenterology. 1973 Jan;64(1):99-105.

引用本文的文献

1
Opportunities in multidimensional trace metal imaging: taking copper-associated disease research to the next level.多维痕量金属成像的机遇:将铜相关疾病研究提升到新的水平。
Anal Bioanal Chem. 2013 Feb;405(6):1809-20. doi: 10.1007/s00216-012-6437-1. Epub 2012 Oct 19.
2
Copper and manganese concentrations in biliary cirrhosis of liver.肝脏胆汁性肝硬化中的铜和锰浓度
Br Med J. 1968 Aug 10;3(5614):344-6. doi: 10.1136/bmj.3.5614.344.
3
Copper kinetics in liver disease.肝脏疾病中的铜动力学
Gut. 1971 Feb;12(2):139-44. doi: 10.1136/gut.12.2.139.
4
Intoxication, detoxication and copper-storage of central nervous tissue at different external Cu(II)-concentrations.不同外部铜(II)浓度下中枢神经组织的中毒、解毒及铜储存
Arch Toxicol. 1974;33(1):17-30. doi: 10.1007/BF00297049.
5
Changes in the distribution of hepatic copper in relation to the progression of Wilson's disease (hepatolenticular degeneration).肝豆状核变性(威尔逊病)进展过程中肝脏铜分布的变化
Am J Pathol. 1968 Dec;53(6):883-901.
6
Biliary copper excretion by hepatocyte lysosomes in the rat. Major excretory pathway in experimental copper overload.大鼠肝细胞溶酶体的胆汁铜排泄。实验性铜过载中的主要排泄途径。
J Clin Invest. 1989 Jan;83(1):30-9. doi: 10.1172/JCI113873.
7
Heavy metal contamination in the river toad, Bufo juxtasper (Inger), near a copper mine in East Malaysia.
Bull Environ Contam Toxicol. 1990 Aug;45(2):272-9. doi: 10.1007/BF01700195.
8
The cornea in normal condition and in Groenouw's macular dystrophy.正常状态下以及格罗诺夫黄斑营养不良状态下的角膜。
Doc Ophthalmol. 1979 Nov 30;47(2):III-VIII, 201-396. doi: 10.1007/BF00568104.