药物诱导的卟啉生物合成。II. 筛选具有卟啉诱导活性药物的简易方法。 - Suppr

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Drug-induced porphyrin biosynthesis. II. Simple procedure for screening drugs for porphyria-inducing activity.

作者信息

Racz W J, Marks G S

出版信息

Biochem Pharmacol. 1969 Aug;18(8):2009-18. doi: 10.1016/0006-2952(69)90298-6.

DOI:10.1016/0006-2952(69)90298-6

PMID:4185885

Abstract

摘要

相似文献

Drug-induced porphyrin biosynthesis. II. Simple procedure for screening drugs for porphyria-inducing activity.药物诱导的卟啉生物合成。II. 筛选具有卟啉诱导活性药物的简易方法。

Biochem Pharmacol. 1969 Aug;18(8):2009-18. doi: 10.1016/0006-2952(69)90298-6.

Drug-induced porphyrin biosynthesis. 3. Inhibition of drug-induced porphyria by protohemin.

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Drug-induced porphyrin biosynthesis. IV. Investigation of the differences in response of isolated liver cells and the liver of the intact chick embryo to porphyria-inducing drugs.

Biochem Pharmacol. 1972 Jan 15;21(2):143-51. doi: 10.1016/0006-2952(72)90264-x.

[Porphyrin synthesis in liver cell culture under the influence of drugs and steroids].

Z Klin Chem Klin Biochem. 1969 Mar;7(2):133-47.

Studies of the relationship between chemical structure and porphyria-inducing activity. IV. Investigations in a cell culture system.

Biochem Pharmacol. 1968 Jul;17(7):1385-99. doi: 10.1016/0006-2952(68)90075-0.

Experimental porphyria. II. Drug-induced porphyrin biosynthesis.实验性卟啉症。II. 药物诱导的卟啉生物合成。

S Afr Med J. 1971 Sep 25:79-81.

Drugs and porphyria.药物与卟啉症

S Afr Med J. 1971 Sep 25:126-33.

Drug-induced porphyrin biosynthesis. VII. Species, sex, and developmental differences in the generation of experimental porphyria.

Can J Physiol Pharmacol. 1972 Jun;50(6):485-9. doi: 10.1139/y72-074.

HEPATOXIC DRUGS CAUSING PORPHYRIA IN MAN AND ANIMALS.导致人和动物患卟啉症的肝毒性药物。

S Afr J Lab Clin Med. 1963 Dec;14:212-20.

Pharmacogenetics and porphyria.药物遗传学与卟啉症

Int Z Klin Pharmakol Ther Toxikol. 1971 Aug;5(1):1-4.

引用本文的文献

Evidence that in chick embryos destruction of hepatic microsomal cytochrome P-450 haem is a general mechanism of induction of delta-aminolaevulinate synthase by porphyria-causing drugs.有证据表明，在鸡胚中，肝微粒体细胞色素P - 450血红素的破坏是卟啉症致病药物诱导δ-氨基乙酰丙酸合酶的一般机制。

Biochem J. 1980 Sep 15;190(3):519-26. doi: 10.1042/bj1900519.

Ferrochelatase and N-alkylated porphyrins.亚铁螯合酶与N-烷基化卟啉

Mol Cell Biochem. 1984 Sep;64(2):127-37. doi: 10.1007/BF00224769.

Studies of the effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin on mammalian hepatic delta-aminolevulinic acid synthetase.2,3,7,8-四氯二苯并-对-二恶英对哺乳动物肝脏δ-氨基乙酰丙酸合成酶影响的研究

Environ Health Perspect. 1973 Sep;5:221-5. doi: 10.1289/ehp.7305221.

[Experimental porphyria. 3. Effects of steroids on latent hexachlorobenzene-porphyria of rats].[实验性卟啉病。3. 类固醇对大鼠潜在性六氯苯-卟啉病的影响]

Arch Dermatol Forsch. 1972;245(4):305-17.

Inhibition of ferrochelatase and accumulation of porphyrins in mouse hepatocyte cultures exposed to porphyrinogenic chemicals.

Arch Toxicol. 1992;66(3):175-81. doi: 10.1007/BF01974011.

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