Drug-induced porphyrin biosynthesis. IV. Investigation of the differences in response of isolated liver cells and the liver of the intact chick embryo to porphyria-inducing drugs.

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作者信息

Racz W J, Marks G S

出版信息

Biochem Pharmacol. 1972 Jan 15;21(2):143-51. doi: 10.1016/0006-2952(72)90264-x.

DOI:10.1016/0006-2952(72)90264-x

PMID:4645552

Abstract

摘要

相似文献

Drug-induced porphyrin biosynthesis. IV. Investigation of the differences in response of isolated liver cells and the liver of the intact chick embryo to porphyria-inducing drugs.

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Drug-induced porphyrin biosynthesis. II. Simple procedure for screening drugs for porphyria-inducing activity.药物诱导的卟啉生物合成。II. 筛选具有卟啉诱导活性药物的简易方法。

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Experimental porphyria. II. Drug-induced porphyrin biosynthesis.实验性卟啉症。II. 药物诱导的卟啉生物合成。

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Cycloheximide enhanced porphyrin synthesis in chick embryo liver: association with an increase in the hepatic glycine pool.放线菌酮增强鸡胚肝脏中的卟啉合成：与肝脏甘氨酸池增加相关。

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Drug induced porphyrin biosynthesis. IX. Levels of 14C-allylisopropylacetamide, 14C-propylisopropylacetamide and metabolite(s) in the livers of chick embryos and mice.

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Drug-induced porphyrin biosynthesis--XIX. Potentiation of the porphyrin-inducing effects of SKF 525-A in the chick embryo liver by 3,5-diethoxycarbonyl-1,4-dihydro-2,4,6-trimethylpyridine, an antihibitor of ferrochelatase.药物诱导的卟啉生物合成——十九。3,5 - 二乙氧羰基 - 1,4 - 二氢 - 2,4,6 - 三甲基吡啶（一种亚铁螯合酶抑制剂）对SKF 525 - A在鸡胚肝脏中诱导卟啉作用的增强。

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Porphyria-inducing activity of a series of pyridine and dihydropyridine compounds. Investigation in a cell culture system.一系列吡啶和二氢吡啶化合物的卟啉症诱导活性。细胞培养系统中的研究。

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Drug-induced porphyrin biosynthesis. VII. Species, sex, and developmental differences in the generation of experimental porphyria.

Can J Physiol Pharmacol. 1972 Jun;50(6):485-9. doi: 10.1139/y72-074.

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Evidence that in chick embryos destruction of hepatic microsomal cytochrome P-450 haem is a general mechanism of induction of delta-aminolaevulinate synthase by porphyria-causing drugs.有证据表明，在鸡胚中，肝微粒体细胞色素P - 450血红素的破坏是卟啉症致病药物诱导δ-氨基乙酰丙酸合酶的一般机制。

Biochem J. 1980 Sep 15;190(3):519-26. doi: 10.1042/bj1900519.

Inhibition of protohaem ferro-lyase in experimental porphyria. Isolation and partial characterization of a modified porphyrin inhibitor.实验性卟啉症中原卟啉铁裂解酶的抑制作用。一种修饰卟啉抑制剂的分离及部分特性鉴定。

Biochem J. 1980 Apr 15;188(1):145-52. doi: 10.1042/bj1880145.

Drug-induced accumulation of uroporphyrin in chicken hepatocyte cultures. Structural requirements for the effect and role of exogenous iron.药物诱导鸡肝细胞培养物中尿卟啉的积累。外源性铁的作用及效应的结构要求。

Biochem J. 1984 Dec 15;224(3):769-77. doi: 10.1042/bj2240769.

Stimulators and inhibitors of hepatic porphyrin formation in human sera.人血清中肝卟啉形成的刺激剂和抑制剂。

J Clin Invest. 1974 Apr;53(4):1167-77. doi: 10.1172/JCI107655.

Decreased liver activity of porphyrin-metal chelatase in hepatic porphyria caused by 3,5-diethoxycarbonyl-1,4-dihydrocollidine. Studies in rats and mice.3,5 - 二乙氧羰基 - 1,4 - 二氢可力丁所致肝性卟啉病中肝脏卟啉 - 金属螯合酶活性降低。大鼠和小鼠研究。

Biochem J. 1973 Jul;134(3):717-27. doi: 10.1042/bj1340717.

Induction of delta-aminolaevulinate synthetase under environmental-stress conditions.环境应激条件下δ-氨基乙酰丙酸合成酶的诱导作用。

Biochem J. 1973 Dec;136(4):1091-6. doi: 10.1042/bj1361091.

Stimulation of the pathway of porphyrin synthesis in the liver of rats and mice by griseofulvin, 3,5-Diethoxycarbonyl-1,4-dihydrocollidine and related drugs: evidence for two basically different mechanisms.灰黄霉素、3,5-二乙氧羰基-1,4-二氢可力丁及相关药物对大鼠和小鼠肝脏卟啉合成途径的刺激作用：两种根本不同机制的证据

Biochem J. 1975 Jan;146(1):285-7. doi: 10.1042/bj1460285.

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