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Shock, cerebral hypoxia, and pulmonary vascular control: the centrineurogenic etiology of the respiratory distress syndrome.

作者信息

Moss G

出版信息

Bull N Y Acad Med. 1973 Aug;49(8):689-93.

PMID:4199642
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1807072/
Abstract
摘要

相似文献

1
Shock, cerebral hypoxia, and pulmonary vascular control: the centrineurogenic etiology of the respiratory distress syndrome.休克、脑缺氧与肺血管控制：呼吸窘迫综合征的中枢神经源性病因
Bull N Y Acad Med. 1973 Aug;49(8):689-93.
2
Cerebral etiology of the respiratory distress syndrome: universal response, with prevention by unilateral pulmonary denervation.呼吸窘迫综合征的脑病因：普遍反应，通过单侧肺去神经支配预防。
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The centrineurogenic etiology of the respiratory distress syndrome: protection by unilateral chronic pulmonary denervation in hemorrhagic shock.呼吸窘迫综合征的中枢神经源性病因：出血性休克中单侧慢性肺去神经支配的保护作用。
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Cerebral etiology of the acute respiratory distress syndrome: diphenylhydantoin prophylaxis.急性呼吸窘迫综合征的脑源性病因：苯妥英预防
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The centrineurogenic etiology of the respiratory distress syndrome: induction by isolated cerebral hypoxemia and prevention by unilateral pulmonary denervation.呼吸窘迫综合征的中枢神经源性病因：由单纯脑缺氧诱导及单侧肺去神经支配预防
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The role of the central nervous system in shock: the centroneurogenic etiology of the respiratory distress syndrome.中枢神经系统在休克中的作用：呼吸窘迫综合征的中枢神经源性病因。
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Cerebral hypotension and shock lung syndrome.脑性低血压和休克肺综合征。
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Cerebral hypoxia as the primary event in the pathogenesis of the "shock lung syndrome".
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Acute interstitial pulmonary fibrosis. Comparative light and electron microscopic study of 19 cases. Pathogenic and therapeutic implications.急性间质性肺纤维化。19例的光镜与电镜对比研究。致病及治疗意义。
Intensive Care Med. 1978 Nov;4(4):181-91. doi: 10.1007/BF01902545.

本文引用的文献

1
EFFECTS OF DIPHENYLHYDANTOIN ON THE DURATION OF RESPIRATORY ACTIVITY DURING ANOXIA.苯妥英对缺氧时呼吸活动持续时间的影响。
J Pharmacol Exp Ther. 1964 Jul;145:34-41.
2
HEMODYNAMICS OF CEREBRAL ARTERIAL ISOLATION.脑动脉隔离的血流动力学
New Physician. 1964 May;13:128-30.
3
CEREBRAL ARTERIAL ISOLATION: THE EFFECTS OF DIFFERENTIAL PRESSURE PERFUSION.脑动脉隔离：压差灌注的影响
J Surg Res. 1964 Apr;4:170-7. doi: 10.1016/s0022-4804(64)80106-2.
4
Effect of elevated left atrial pressure and decreased plasma protein concentration on the development of pulmonary edema.左心房压力升高和血浆蛋白浓度降低对肺水肿发展的影响。
Circ Res. 1959 Jul;7(4):649-57. doi: 10.1161/01.res.7.4.649.
5
Pressure changes in the dog lung secondary to hemorrhagic shock: protective effect of pulmonary reimplantation.失血性休克继发犬肺内的压力变化：肺再植入的保护作用
Ann Surg. 1969 Apr;169(4):592-8. doi: 10.1097/00000658-196904000-00016.
6
Oxygen tension in the cerebral cortex of dogs in haemorrhagic shock and its change after oxygen therapy.失血性休克犬大脑皮质的氧张力及其氧疗后的变化。
Acta Anaesthesiol Scand. 1968;12(4):171-82. doi: 10.1111/j.1399-6576.1968.tb00421.x.
7
Regional blood flow in hemorrhagic shock by the distribution of labeled microspheres.
Surg Forum. 1968;19:14-5.
8
Prevention of lesions of the lung secondary to hemorrhagic shock.预防出血性休克继发的肺部损伤。
Surg Gynecol Obstet. 1968 Nov;127(5):1005-10.
9
Changes in composition of cerebrospinal fluid in hemorrhagic shock.失血性休克时脑脊液成分的变化
Surg Forum. 1970;21:53-5.
10
Pulmonary microcirculation in hemorrhagic shock.失血性休克中的肺微循环。
Surg Forum. 1970;21:25-7.