Moss G, Stein A A
J Trauma. 1976 May;16(5):361-4.
Anemic beagles were subjected to 40 mm Hg hemorrhagic shock for 2 hours, which uniformly induced the pulmonary lesions of the "respiratory distress syndrome" (RDS) bilaterally in all six controls. For six subjects with complete denervation of the left lung 2 months previously, the shock regimen induced the lesions in the normally innervated right lungs; all reimplanted, denervated left lungs remained anatomically intact. This is presented as additional evidence for a centrineurogenic etiology for RDS.
贫血的比格犬接受40毫米汞柱的失血性休克2小时，这在所有六只对照犬中均双侧均匀诱发了“呼吸窘迫综合征”（RDS）的肺部病变。对于2个月前左肺完全去神经支配的六只实验对象，休克方案在正常神经支配的右肺诱发了病变；所有重新植入的、去神经支配的左肺在解剖学上保持完整。这被作为RDS中枢神经源性病因的额外证据呈现。
