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Tritiated thymidine incorporation into aortic cells in vivo: cell regeneration in spontaneous atherosclerosis in monkeys.

作者信息

Murata K

出版信息

Experientia. 1967 Sep 15;23(9):732-3. doi: 10.1007/BF02154143.

DOI:10.1007/BF02154143
PMID:4228897
Abstract
摘要

相似文献

1
Tritiated thymidine incorporation into aortic cells in vivo: cell regeneration in spontaneous atherosclerosis in monkeys.
Experientia. 1967 Sep 15;23(9):732-3. doi: 10.1007/BF02154143.
2
The in vitro incorporation of ( 3 H)thymidine into DNA and of 32 P into phospholipids and RNA in the aorta of Rhesus monkeys during early atherogenesis.恒河猴早期动脉粥样硬化形成过程中，（3H）胸腺嘧啶核苷在其主动脉DNA中的体外掺入以及32P在磷脂和RNA中的体外掺入。
Atherosclerosis. 1973 Mar-Apr;17(2):269-80. doi: 10.1016/0021-9150(73)90093-2.
3
Use of aortic medial explants in the study of atherosclerosis.主动脉中膜外植体在动脉粥样硬化研究中的应用。
Exp Mol Pathol. 1973 Apr;18(2):177-89. doi: 10.1016/0014-4800(73)90015-4.
4
Incorporation of labelled sulphate into aortic tissue during ageing.衰老过程中标记硫酸盐在主动脉组织中的掺入。
Physiol Bohemoslov. 1973;22(2):143-5.
5
RADIOAUTOGRAPHIC STUDIES OF THE PROLIFERATIVE PHASE OF PRIMATE ATHEROGENESIS.
Surg Forum. 1963;14:306-7.
6
[Changes in the viscosity of mucopolysaccharides of the human aorta in calcification of the arteries].[动脉钙化时人主动脉黏多糖黏度的变化]
Dtsch Z Verdau Stoffwechselkr. 1972;32(5):355-9.
7
Aortic total glycosaminoglycan and dermatan sulfate changes in atherosclerotic rhesus monkeys.动脉粥样硬化恒河猴的主动脉总糖胺聚糖和硫酸皮肤素变化
Lab Invest. 1978 Oct;39(4):322-8.
8
Preliminary experience with mitotic activity of cellular elements in the atherosclerotic plaques of cholesterol-fed rabbits studied by labeling with tritiated thymidine.通过用氚标记的胸腺嘧啶核苷进行标记研究胆固醇喂养兔动脉粥样硬化斑块中细胞成分有丝分裂活性的初步经验。
Ann N Y Acad Sci. 1968 Nov 21;149(2):699-709. doi: 10.1111/j.1749-6632.1968.tb53829.x.
9
[3H-thymidine incorporation into human aorta cells in primary culture. An autoradiographic study].[3H-胸腺嘧啶核苷掺入原代培养的人主动脉细胞。一项放射自显影研究]
Tsitologiia. 1983 Jul;25(7):849-51.
10
[Mucopolysaccharides of the aortic arch during aging].
G Gerontol. 1962 Feb;10:189-99.

引用本文的文献

1
Synthesis and transport of cholesterol in aortic cells: radiosiotopic studies in cell culture.
Experientia. 1968 Nov 15;24(11):1111-2. doi: 10.1007/BF02147788.
2
Effect of prednisone on the noradrenaline-indeced cell proliferation in the arterial wall.泼尼松对去甲肾上腺素诱导的动脉壁细胞增殖的影响。
Experientia. 1973 Jun 15;29(6):703-4. doi: 10.1007/BF01944786.

本文引用的文献

1
A method for the determination of desoxyribonucleic acid, ribonucleic acid, and phosphoproteins in animal tissues.一种测定动物组织中脱氧核糖核酸、核糖核酸和磷蛋白的方法。
J Biol Chem. 1945;161:83-9.
2
The effects of sulfated polysaccharides obtained from seaweeds on experimental atherosclerosis.从海藻中提取的硫酸化多糖对实验性动脉粥样硬化的影响。
J Gerontol. 1962 Jan;17:30-6. doi: 10.1093/geronj/17.1.30.
3
Further studies on the inhibitory effects of synthetic sulfated monosaccharides on experimental atherosclerosis.合成硫酸化单糖对实验性动脉粥样硬化抑制作用的进一步研究。
Jpn Heart J. 1961 Oct;2:493-504. doi: 10.1536/ihj.2.493.
4
THE INFLUENCE OF AGE AND ATHEROSCLEROSIS ON THE CHEMISTRY OF AORTIC INTIMA.2. COLLAGEN AND MUCOPOLYSACCHARIDES.年龄和动脉粥样硬化对主动脉内膜化学组成的影响。2. 胶原蛋白和粘多糖
J Atheroscler Res. 1965 Mar-Apr;5(2):241-8. doi: 10.1016/s0368-1319(65)80065-5.
5
INCORPORATION OF THYMIDINE INTO DNA OF MOUSE ORGANS.胸腺嘧啶核苷掺入小鼠器官的DNA中。
Arch Pathol. 1964 Sep;78:245-53.
6
TRITIATED THYMIDINE UPTAKE IN RAT HYPERTENSION.大鼠高血压中氚标记胸腺嘧啶核苷的摄取
Arch Pathol. 1964 Sep;78:209-21.
7
ATHEROSCLEROSIS IN THE SQUIRREL MONKEY. NATURALLY OCCURRING LESIONS OF THE AORTA AND CORONARY ARTERIES.
Arch Pathol. 1964 Jul;78:16-23.
8
ACID MUCOPOLYSACCHARIDES IN HUMAN AORTIC TISSUE: THEIR DISTRIBUTION AT DIFFERENT STAGES OF ATHEROSCLEROSIS.人主动脉组织中的酸性粘多糖：它们在动脉粥样硬化不同阶段的分布
Lancet. 1963 Aug 31;2(7305):439-40. doi: 10.1016/s0140-6736(63)92179-2.
9
Role of hyperplasia in vascular lesions of cholesterol-fed rabbits studied with thymidine-H3 autoradiography.用氚标记胸腺嘧啶核苷放射自显影术研究增生在喂胆固醇家兔血管病变中的作用。
Circ Res. 1962 Aug;11:329-36. doi: 10.1161/01.res.11.2.329.
10
Inhibitory effect of sulfated mono- and polysaccharides on experimental hyperlipemia and atherosclerosis in rabbits.硫酸化单糖和多糖对兔实验性高脂血症和动脉粥样硬化的抑制作用。
Jpn Heart J. 1961 Apr;2:198-209. doi: 10.1536/ihj.2.198.